Chunyan Gao

Daqing, China

Last Updated: 09/26/2026

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Found 12 publications

Red blood cell-derived microparticles induce kidney injury by triggering endothelial cell ferroptosis in intravascular hemolysis.

Red blood cell-derived microparticles induce kidney injury by triggering endothelial cell ferroptosis in intravascular hemolysis.

Journal: Redox biology

Published: July 12, 2025

NRF2/miR-17-5p/TMEM16F axis regulates the crosstalk of inflammation and thrombosis in sepsis.

NRF2/miR-17-5p/TMEM16F axis regulates the crosstalk of inflammation and thrombosis in sepsis.

Journal: Journal of thrombosis and haemostasis : JTH

Published: April 13, 2025

Pan-Cancer Analysis of ANO6 and Experimental Validation in Metastatic Melanoma.

Pan-Cancer Analysis of ANO6 and Experimental Validation in Metastatic Melanoma.

Journal: Biochemical genetics

Published: November 05, 2024

Erythrophagocytosis-induced ferroptosis contributes to pulmonary microvascular thrombosis and thrombotic vascular remodeling in pulmonary arterial hypertension.

Erythrophagocytosis-induced ferroptosis contributes to pulmonary microvascular thrombosis and thrombotic vascular remodeling in pulmonary arterial hypertension.

Journal: Journal of thrombosis and haemostasis : JTH

Published: March 18, 2024

Ferroptosis of Endothelial Cells Triggered by Erythrophagocytosis Contributes to Thrombogenesis in Uremia.

Ferroptosis of Endothelial Cells Triggered by Erythrophagocytosis Contributes to Thrombogenesis in Uremia.

Journal: Thrombosis and haemostasis

Published: June 26, 2023

TMEM16F mediated phosphatidylserine exposure and microparticle release on erythrocyte contribute to hypercoagulable state in hyperuricemia.

TMEM16F mediated phosphatidylserine exposure and microparticle release on erythrocyte contribute to hypercoagulable state in hyperuricemia.

Journal: Blood cells, molecules & diseases

Published: March 08, 2022

Publisher Correction: Phosphatidylserine-mediated platelet clearance by endothelium decreases platelet aggregates and procoagulant activity in sepsis.

Publisher Correction: Phosphatidylserine-mediated platelet clearance by endothelium decreases platelet aggregates and procoagulant activity in sepsis.

Journal: Scientific reports

Published: April 15, 2018

Phosphatidylserine-mediated platelet clearance by endothelium decreases platelet aggregates and procoagulant activity in sepsis.

Phosphatidylserine-mediated platelet clearance by endothelium decreases platelet aggregates and procoagulant activity in sepsis.

Journal: Scientific reports

Published: April 01, 2016

Indolic uremic solutes enhance procoagulant activity of red blood cells through phosphatidylserine exposure and microparticle release.

Indolic uremic solutes enhance procoagulant activity of red blood cells through phosphatidylserine exposure and microparticle release.

Journal: Toxins

Published: July 28, 2015

Thrombotic Role of Blood and Endothelial Cells in Uremia through Phosphatidylserine Exposure and Microparticle Release.

Thrombotic Role of Blood and Endothelial Cells in Uremia through Phosphatidylserine Exposure and Microparticle Release.

Journal: PloS one

Published: May 20, 2015

Endothelial cell phagocytosis of senescent neutrophils decreases procoagulant activity.

Endothelial cell phagocytosis of senescent neutrophils decreases procoagulant activity.

Journal: Thrombosis and haemostasis

Published: December 05, 2012

Role of erythrocytes and platelets in the hypercoagulable status in polycythemia vera through phosphatidylserine exposure and microparticle generation.

Role of erythrocytes and platelets in the hypercoagulable status in polycythemia vera through phosphatidylserine exposure and microparticle generation.

Journal: Thrombosis and haemostasis

Published: November 09, 2012

Showing 1-12 of 12

Last Updated: 09/26/2026