Claudia Meyer
Mannheim, BW, Germany
Last Updated: 09/26/2026
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Found 16 publications
Csnk1a1 E98 mutations rewire signaling and metabolism in del(5q) myelodysplastic neoplasms.
Journal: Leukemia
Published: April 24, 2026
Publisher Correction: Multiple oestradiol functions inhibit ferroptosis and acute kidney injury.
Journal: Nature
Published: January 21, 2026
Publisher Correction: Multiple oestradiol functions inhibit ferroptosis and acute kidney injury.
Journal: Nature
Published: August 26, 2025
Galectic Control of Ferroptosis?
Journal: Journal of the American Society of Nephrology : JASN
Published: June 10, 2025
Treatment with siRNAs is commonly associated with GPX4 up-regulation and target knockdown-independent sensitization to ferroptosis.
Journal: Science advances
Published: March 15, 2024
Multiple oestradiol functions inhibit ferroptosis and acute kidney injury.
Journal: Nature
Published: November 12, 2023
Gasdermin D-deficient mice are hypersensitive to acute kidney injury.
Journal: Cell death & disease
Published: July 03, 2022
Dexamethasone sensitizes to ferroptosis by glucocorticoid receptor-induced dipeptidase-1 expression and glutathione depletion.
Journal: Science advances
Published: February 02, 2022
Rubicon-deficiency sensitizes mice to mixed lineage kinase domain-like (MLKL)-mediated kidney ischemia-reperfusion injury.
Journal: Cell death & disease
Published: January 13, 2022
Mechanisms and Models of Kidney Tubular Necrosis and Nephron Loss.
Journal: Journal of the American Society of Nephrology : JASN
Published: January 13, 2022
Dysfunction of the key ferroptosis-surveilling systems hypersensitizes mice to tubular necrosis during acute kidney injury.
Journal: Nature communications
Published: March 19, 2021
Anti-ferroptotic mechanism of IL4i1-mediated amino acid metabolism.
Journal: eLife
Published: November 11, 2020
Last Updated: 09/26/2026