Claudio A. Masuda
Rio De Janeiro, RJ, Brazil
Last Updated: 09/26/2026
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Found 18 publications
Galactose-1-phosphate inhibits cytochrome c oxidase and causes mitochondrial dysfunction in classic galactosemia.
Journal: Biochimica et biophysica acta. Molecular basis of disease
Published: March 05, 2024
Author Correction: Apoptotic stress causes mtDNA release during senescence and drives the SASP.
Journal: Nature
Published: January 03, 2024
Apoptotic stress causes mtDNA release during senescence and drives the SASP.
Journal: Nature
Published: January 10, 2022
Sphingolipid depletion suppresses UPR activation and promotes galactose hypersensitivity in yeast models of classic galactosemia.
Journal: Biochimica et biophysica acta. Molecular basis of disease
Published: August 19, 2021
Rqc1 and other yeast proteins containing highly positively charged sequences are not targets of the RQC complex.
Journal: The Journal of biological chemistry
Published: September 21, 2020
Neutrophils induce paracrine telomere dysfunction and senescence in ROS-dependent manner.
Journal: The EMBO journal
Published: June 24, 2020
Regulation of sphingolipid synthesis by the G1/S transcription factor Swi4.
Journal: Biochimica et biophysica acta. Molecular and cell biology of lipids
Published: May 11, 2020
The yeast protein Ubx4p contributes to mitochondrial respiration and lithium-galactose-mediated activation of the unfolded protein response.
Journal: The Journal of biological chemistry
Published: October 09, 2019
Methyl and Ethylmercury elicit oxidative stress and unbalance the antioxidant system in Saccharomyces cerevisiae.
Journal: Chemico-biological interactions
Published: June 05, 2019
Induction of triacylglycerol synthesis in yeast by cell cycle arrest.
Journal: FEMS yeast research
Published: November 13, 2018
The galactose-induced decrease in phosphate levels leads to toxicity in yeast models of galactosemia.
Journal: Biochimica et biophysica acta. Molecular basis of disease
Published: November 09, 2016
Last Updated: 09/26/2026