Colleen A. Flanagan

7 York Road, Parktown
Johannesburg, GT 2193, South Africa

Last Updated: 09/26/2026

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Found 13 publications

Tumor Necrosis Factor-α Mediates Inflammation-induced Early-Stage Left Ventricular Systolic Dysfunction.

Tumor Necrosis Factor-α Mediates Inflammation-induced Early-Stage Left Ventricular Systolic Dysfunction.

Journal: Journal of cardiovascular pharmacology

Published: September 24, 2022

Increased protein phosphatase 5 expression in inflammation-induced left ventricular dysfunction in rats.

Increased protein phosphatase 5 expression in inflammation-induced left ventricular dysfunction in rats.

Journal: BMC cardiovascular disorders

Published: April 28, 2022

Inflammation-induced left ventricular fibrosis is partially mediated by tumor necrosis factor-α.

Inflammation-induced left ventricular fibrosis is partially mediated by tumor necrosis factor-α.

Journal: Physiological reports

Published: July 09, 2021

Genetics of ncHH: from a peculiar inheritance of a novel GNRHR mutation to a comprehensive review of the literature.

Genetics of ncHH: from a peculiar inheritance of a novel GNRHR mutation to a comprehensive review of the literature.

Journal: Andrology

Published: June 29, 2018

Glu2.53(90) of the GnRH receptor is part of the conserved G protein-coupled receptor structure and does not form a salt-bridge with Lys3.32(121).

Glu2.53(90) of the GnRH receptor is part of the conserved G protein-coupled receptor structure and does not form a salt-bridge with Lys3.32(121).

Journal: Molecular and cellular endocrinology

Published: June 26, 2018

Similarities and differences in the reproductive phenotypes of women with congenital hypogonadotrophic hypogonadism caused by GNRHR mutations and women with polycystic ovary syndrome.

Similarities and differences in the reproductive phenotypes of women with congenital hypogonadotrophic hypogonadism caused by GNRHR mutations and women with polycystic ovary syndrome.

Journal: Human reproduction (Oxford, England)

Published: March 31, 2018

Gonadotropin-Releasing Hormone (GnRH) Receptor Structure and GnRH Binding.

Gonadotropin-Releasing Hormone (GnRH) Receptor Structure and GnRH Binding.

Journal: Frontiers in endocrinology

Published: August 08, 2017

GPCR-radioligand binding assays.

GPCR-radioligand binding assays.

Journal: Methods in cell biology

Published: March 02, 2016

The carboxy-terminal tail or the intracellular loop 3 is required for β-arrestin-dependent internalization of a mammalian type II GnRH receptor.

The carboxy-terminal tail or the intracellular loop 3 is required for β-arrestin-dependent internalization of a mammalian type II GnRH receptor.

Journal: Molecular and cellular endocrinology

Published: December 31, 2014

Histidine(7.36(305)) in the conserved peptide receptor activation domain of the gonadotropin releasing hormone receptor couples peptide binding and receptor activation.

Histidine(7.36(305)) in the conserved peptide receptor activation domain of the gonadotropin releasing hormone receptor couples peptide binding and receptor activation.

Journal: Molecular and cellular endocrinology

Published: August 20, 2014

Receptor conformation and constitutive activity in CCR5 chemokine receptor function and HIV infection.

Receptor conformation and constitutive activity in CCR5 chemokine receptor function and HIV infection.

Journal: Advances in pharmacology (San Diego, Calif.)

Published: June 17, 2014

R31C GNRH1 mutation and congenital hypogonadotropic hypogonadism.

R31C GNRH1 mutation and congenital hypogonadotropic hypogonadism.

Journal: PloS one

Published: February 11, 2013

Showing 1-12 of 13

Last Updated: 09/26/2026