Conor M. Henry

Dublin, L, Ireland

Last Updated: 09/26/2026

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Found 14 publications

TRAIL Receptors Serve as Stress-Associated Molecular Patterns to Promote ER-Stress-Induced Inflammation.

TRAIL Receptors Serve as Stress-Associated Molecular Patterns to Promote ER-Stress-Induced Inflammation.

Journal: Developmental cell
Published: May 30, 2019

Suppressing IL-36-driven inflammation using peptide pseudosubstrates for neutrophil proteases.

Suppressing IL-36-driven inflammation using peptide pseudosubstrates for neutrophil proteases.

Journal: Cell death & disease
Published: November 28, 2017

Identification of small-molecule elastase inhibitors as antagonists of IL-36 cytokine activation.

Identification of small-molecule elastase inhibitors as antagonists of IL-36 cytokine activation.

Journal: FEBS open bio
Published: November 09, 2017

Neutrophil extracellular traps can serve as platforms for processing and activation of IL-1 family cytokines.

Neutrophil extracellular traps can serve as platforms for processing and activation of IL-1 family cytokines.

Journal: The FEBS journal
Published: December 02, 2016

Extracellular Neutrophil Proteases Are Efficient Regulators of IL-1, IL-33, and IL-36 Cytokine Activity but Poor Effectors of Microbial Killing.

Extracellular Neutrophil Proteases Are Efficient Regulators of IL-1, IL-33, and IL-36 Cytokine Activity but Poor Effectors of Microbial Killing.

Journal: Cell reports
Published: September 21, 2016

Caspase-8 Acts in a Non-enzymatic Role as a Scaffold for Assembly of a Pro-inflammatory "FADDosome" Complex upon TRAIL Stimulation.

Caspase-8 Acts in a Non-enzymatic Role as a Scaffold for Assembly of a Pro-inflammatory "FADDosome" Complex upon TRAIL Stimulation.

Journal: Molecular cell
Published: July 19, 2016

Production of biologically active IL-36 family cytokines through insertion of N-terminal caspase cleavage motifs.

Production of biologically active IL-36 family cytokines through insertion of N-terminal caspase cleavage motifs.

Journal: FEBS open bio
Published: December 04, 2015

Neutrophil-Derived Proteases Escalate Inflammation through Activation of IL-36 Family Cytokines.

Neutrophil-Derived Proteases Escalate Inflammation through Activation of IL-36 Family Cytokines.

Journal: Cell reports
Published: February 23, 2015

Necroptosis suppresses inflammation via termination of TNF- or LPS-induced cytokine and chemokine production.

Necroptosis suppresses inflammation via termination of TNF- or LPS-induced cytokine and chemokine production.

Journal: Cell death and differentiation
Published: September 08, 2014

Distinguishing between apoptosis, necrosis, necroptosis and other cell death modalities.

Distinguishing between apoptosis, necrosis, necroptosis and other cell death modalities.

Journal: Methods (San Diego, Calif.)
Published: June 18, 2013

Measuring apoptosis by microscopy and flow cytometry.

Measuring apoptosis by microscopy and flow cytometry.

Journal: Methods (San Diego, Calif.)
Published: October 12, 2012

A perspective on mammalian caspases as positive and negative regulators of inflammation.

A perspective on mammalian caspases as positive and negative regulators of inflammation.

Journal: Molecular cell
Published: May 29, 2012
Showing 1-12 of 14

Last Updated: 09/26/2026