Coral Ampurdanes

Barcelona, CT, Spain

Last Updated: 09/26/2026

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Found 10 publications

PARP2 deficiency impairs pancreatic cancer progression by promoting genomic instability and antitumor immunity.

PARP2 deficiency impairs pancreatic cancer progression by promoting genomic instability and antitumor immunity.

Journal: Science advances
Published: May 08, 2026

Attenuation of Tumor Burden in Response to Rucaparib in Lung Adenocarcinoma: The Contribution of Oxidative Stress, Apoptosis, and DNA Damage.

Attenuation of Tumor Burden in Response to Rucaparib in Lung Adenocarcinoma: The Contribution of Oxidative Stress, Apoptosis, and DNA Damage.

Journal: International journal of molecular sciences
Published: November 15, 2022

Distinct roles for PARP-1 and PARP-2 in c-Myc-driven B-cell lymphoma in mice.

Distinct roles for PARP-1 and PARP-2 in c-Myc-driven B-cell lymphoma in mice.

Journal: Blood
Published: June 07, 2021

Increased PARP Activity and DNA Damage in NSCLC Patients: The Influence of COPD.

Increased PARP Activity and DNA Damage in NSCLC Patients: The Influence of COPD.

Journal: Cancers
Published: October 22, 2020

Coordinated signals from PARP-1 and PARP-2 are required to establish a proper T cell immune response to breast tumors in mice.

Coordinated signals from PARP-1 and PARP-2 are required to establish a proper T cell immune response to breast tumors in mice.

Journal: Oncogene
Published: September 10, 2019

Coordinated signals from the DNA repair enzymes PARP-1 and PARP-2 promotes B-cell development and function.

Coordinated signals from the DNA repair enzymes PARP-1 and PARP-2 promotes B-cell development and function.

Journal: Cell death and differentiation
Published: September 27, 2018

PARP-1/PARP-2 double deficiency in mouse T cells results in faulty immune responses and T lymphomas.

PARP-1/PARP-2 double deficiency in mouse T cells results in faulty immune responses and T lymphomas.

Journal: Scientific reports
Published: August 23, 2016

PARP-2 sustains erythropoiesis in mice by limiting replicative stress in erythroid progenitors.

PARP-2 sustains erythropoiesis in mice by limiting replicative stress in erythroid progenitors.

Journal: Cell death and differentiation
Published: June 27, 2014

Parp-2 is required to maintain hematopoiesis following sublethal γ-irradiation in mice.

Parp-2 is required to maintain hematopoiesis following sublethal γ-irradiation in mice.

Journal: Blood
Published: May 17, 2013

Tissue plasminogen activator induces microglial inflammation via a noncatalytic molecular mechanism involving activation of mitogen-activated protein kinases and Akt signaling pathways and AnnexinA2 and Galectin-1 receptors.

Tissue plasminogen activator induces microglial inflammation via a noncatalytic molecular mechanism involving activation of mitogen-activated protein kinases and Akt signaling pathways and AnnexinA2 and Galectin-1 receptors.

Journal: Glia
Published: November 22, 2011
Showing 1-10 of 10

Last Updated: 09/26/2026