Cristina Silva-Ribeiro
Rotterdam, ZH, Netherlands
Last Updated: 09/26/2026
Save publications for later
Sign Up
Not sure about your diagnosis?
Check Your Symptoms
Found 11 publications
Recruitment and release of XPG during NER is controlled by pre- and post-incision factors and EXO1.
Journal: The Journal of cell biology
Published: February 20, 2026
Persistent TFIIH binding to non-excised DNA damage causes cell and developmental failure.
Journal: Nature communications
Published: April 22, 2023
Detection of oxaliplatin- and cisplatin-DNA lesions requires different global genome repair mechanisms that affect their clinical efficacy.
Journal: NAR cancer
Published: March 23, 2023
Different SWI/SNF complexes coordinately promote R-loop- and RAD52-dependent transcription-coupled homologous recombination.
Journal: Nucleic acids research
Published: November 09, 2022
XPG: a multitasking genome caretaker.
Journal: Cellular and molecular life sciences : CMLS
Published: November 26, 2021
Ubiquitin and TFIIH-stimulated DDB2 dissociation drives DNA damage handover in nucleotide excision repair.
Journal: Nature communications
Published: December 20, 2019
SWI/SNF: Complex complexes in genome stability and cancer.
Journal: DNA repair
Published: December 07, 2018
Base and nucleotide excision repair facilitate resolution of platinum drugs-induced transcription blockage.
Journal: Nucleic acids research
Published: June 14, 2018
Repair protein persistence at DNA lesions characterizes XPF defect with Cockayne syndrome features.
Journal: Nucleic acids research
Published: March 29, 2018
DNA damage sensitivity of SWI/SNF-deficient cells depends on TFIIH subunit p62/GTF2H1.
Journal: Nature communications
Published: January 08, 2018
Showing 1-11 of 11
Last Updated: 09/26/2026