Cyril J. Jagaraj
75 Talavera Road
Sydney, NSW 2109, Australia
Sydney, NSW 2109, Australia
Last Updated: 09/26/2026
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Found 16 publications
Cofilin hyperphosphorylation triggers TDP-43 pathology in sporadic amyotrophic lateral sclerosis.
Journal: Brain : a journal of neurology
Published: July 31, 2025
A Slower-Progressing TDP-43 rNLS8 Mouse Model for ALS: Implications for Preclinical and Mechanistic Studies.
Journal: Neuromolecular medicine
Published: May 08, 2025
IL-6 trans-signalling is elevated in ALS models and drives TDP-43 induced inflammatory responses in microglia.
Journal: Brain, behavior, and immunity
Published: December 12, 2024
Molecular hallmarks of ageing in amyotrophic lateral sclerosis.
Journal: Cellular and molecular life sciences : CMLS
Published: December 05, 2023
Protein Disulfide Isomerase Endoplasmic Reticulum Protein 57 (ERp57) is Protective Against ALS-Associated Mutant TDP-43 in Neuronal Cells.
Journal: Neuromolecular medicine
Published: November 16, 2023
C9orf72-Associated Dipeptide Repeat Expansions Perturb ER-Golgi Vesicular Trafficking, Inducing Golgi Fragmentation and ER Stress, in ALS/FTD.
Journal: Molecular neurobiology
Published: December 22, 2022
TDP-43 is a ubiquitylation substrate of the SCFcyclin F complex.
Journal: Neurobiology of disease
Published: October 26, 2021
Emerging Evidence Highlighting the Importance of Redox Dysregulation in the Pathogenesis of Amyotrophic Lateral Sclerosis (ALS).
Journal: Frontiers in cellular neuroscience
Published: July 14, 2020
Impaired NHEJ repair in amyotrophic lateral sclerosis is associated with TDP-43 mutations.
Journal: Molecular neurodegeneration
Published: October 20, 2019
Genetic and immunopathological analysis of CHCHD10 in Australian amyotrophic lateral sclerosis and frontotemporal dementia and transgenic TDP-43 mice.
Journal: Journal of neurology, neurosurgery, and psychiatry
Published: August 05, 2019
Last Updated: 09/26/2026