Damian Krzyzanowski

Lodowa Street 102
Lodz, LD 93232, Poland

Last Updated: 09/26/2026

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Found 9 publications

The paradox of autoimmunity and autoinflammation in inherited neutrophil disorders - in search of common patterns.

The paradox of autoimmunity and autoinflammation in inherited neutrophil disorders - in search of common patterns.

Journal: Frontiers in immunology

Published: December 20, 2022

Differential Action of Silver Nanoparticles on ABCB1 (MDR1) and ABCC1 (MRP1) Activity in Mammalian Cell Lines.

Differential Action of Silver Nanoparticles on ABCB1 (MDR1) and ABCC1 (MRP1) Activity in Mammalian Cell Lines.

Journal: Materials (Basel, Switzerland)

Published: May 24, 2021

Neutrophil Elastase Defects in Congenital Neutropenia.

Neutrophil Elastase Defects in Congenital Neutropenia.

Journal: Frontiers in immunology

Published: January 15, 2021

Snail Overexpression Alters the microRNA Content of Extracellular Vesicles Released from HT29 Colorectal Cancer Cells and Activates Pro-Inflammatory State In Vivo.

Snail Overexpression Alters the microRNA Content of Extracellular Vesicles Released from HT29 Colorectal Cancer Cells and Activates Pro-Inflammatory State In Vivo.

Journal: Cancers

Published: October 09, 2020

Glypican-1 Level Is Elevated in Extracellular Vesicles Released from MC38 Colon Adenocarcinoma Cells Overexpressing Snail.

Glypican-1 Level Is Elevated in Extracellular Vesicles Released from MC38 Colon Adenocarcinoma Cells Overexpressing Snail.

Journal: Cells

Published: May 29, 2020

Adenosine Analogues as Opposite Modulators of the Cisplatin Resistance of Ovarian Cancer Cells.

Adenosine Analogues as Opposite Modulators of the Cisplatin Resistance of Ovarian Cancer Cells.

Journal: Anti-cancer agents in medicinal chemistry

Published: October 15, 2018

Cisplatin-induced ERK1/2 activity promotes G1 to S phase progression which leads to chemoresistance of ovarian cancer cells.

Cisplatin-induced ERK1/2 activity promotes G1 to S phase progression which leads to chemoresistance of ovarian cancer cells.

Journal: Oncotarget

Published: July 26, 2017

Glucose availability determines silver nanoparticles toxicity in HepG2.

Glucose availability determines silver nanoparticles toxicity in HepG2.

Journal: Journal of nanobiotechnology

Published: July 16, 2015

Collateral sensitivity: ABCG2-overexpressing cells are more vulnerable to oxidative stress.

Collateral sensitivity: ABCG2-overexpressing cells are more vulnerable to oxidative stress.

Journal: Free radical biology & medicine

Published: January 24, 2014

Showing 1-9 of 9

Last Updated: 09/26/2026