Dana Hodroj

Montpellier, France

Last Updated: 09/26/2026

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Found 8 publications

MCM8- and MCM9 Deficiencies Cause Lifelong Increased Hematopoietic DNA Damage Driving p53-Dependent Myeloid Tumors.

MCM8- and MCM9 Deficiencies Cause Lifelong Increased Hematopoietic DNA Damage Driving p53-Dependent Myeloid Tumors.

Journal: Cell reports
Published: December 19, 2025

Shining light on drug discovery: optogenetic screening for TopBP1 biomolecular condensate inhibitors.

Shining light on drug discovery: optogenetic screening for TopBP1 biomolecular condensate inhibitors.

Journal: NAR cancer
Published: June 04, 2025

Dihydropyrimidinase protects from DNA replication stress caused by cytotoxic metabolites.

Dihydropyrimidinase protects from DNA replication stress caused by cytotoxic metabolites.

Journal: Nucleic acids research
Published: April 17, 2019

MCM8- and MCM9 Deficiencies Cause Lifelong Increased Hematopoietic DNA Damage Driving p53-Dependent Myeloid Tumors.

MCM8- and MCM9 Deficiencies Cause Lifelong Increased Hematopoietic DNA Damage Driving p53-Dependent Myeloid Tumors.

Journal: Cell reports
Published: January 09, 2019

Ddx19 links mRNA nuclear export with progression of transcription and replication and suppresses genomic instability upon DNA damage in proliferating cells.

Ddx19 links mRNA nuclear export with progression of transcription and replication and suppresses genomic instability upon DNA damage in proliferating cells.

Journal: Nucleus (Austin, Tex.)
Published: July 12, 2017

An ATR-dependent function for the Ddx19 RNA helicase in nuclear R-loop metabolism.

An ATR-dependent function for the Ddx19 RNA helicase in nuclear R-loop metabolism.

Journal: The EMBO journal
Published: June 29, 2016

PIP degron proteins, substrates of CRL4Cdt2, and not PIP boxes, interfere with DNA polymerase η and κ focus formation on UV damage.

PIP degron proteins, substrates of CRL4Cdt2, and not PIP boxes, interfere with DNA polymerase η and κ focus formation on UV damage.

Journal: Nucleic acids research
Published: January 16, 2014

Combination of arsenic and interferon-α inhibits expression of KSHV latent transcripts and synergistically improves survival of mice with primary effusion lymphomas.

Combination of arsenic and interferon-α inhibits expression of KSHV latent transcripts and synergistically improves survival of mice with primary effusion lymphomas.

Journal: PloS one
Published: April 23, 2013
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Last Updated: 09/26/2026