Daniel Ruhnau
Vienna, 1210, Austria
Last Updated: 09/26/2026
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Found 9 publications
Exposure of broiler chickens to deoxynivalenol and Campylobacter jejuni induces substantial changes in intestinal gene expression.
Journal: Scientific reports
Published: September 17, 2024
Diametral influence of deoxynivalenol (DON) and deepoxy-deoxynivalenol (DOM-1) on the growth of Campylobacter jejuni with consequences on the bacterial transcriptome.
Journal: BMC microbiology
Published: November 13, 2023
Paracellular intestinal permeability of chickens induced by DON and/or C. jejuni is associated with alterations in tight junction mRNA expression.
Journal: Microbial pathogenesis
Published: October 14, 2021
Deepoxy-deoxynivalenol (DOM-1), a derivate of deoxynivalenol (DON), exhibits less toxicity on intestinal barrier function, Campylobacter jejuni colonization and translocation in broiler chickens.
Journal: Gut pathogens
Published: January 15, 2021
The Mycotoxin Deoxynivalenol (DON) Promotes Campylobacter jejuni Multiplication in the Intestine of Broiler Chickens With Consequences on Bacterial Translocation and Gut Integrity.
Journal: Frontiers in veterinary science
Published: June 18, 2020
Feeding of deoxynivalenol increases the intestinal paracellular permeability of broiler chickens.
Journal: Archives of toxicology
Published: February 20, 2019
Presence of Avibacterium paragallinarum and Histopathologic Lesions Corresponds with Clinical Signs in a Co-infection Model with Gallibacterium anatis.
Journal: Avian diseases
Published: September 29, 2017
Showing 1-9 of 9
Last Updated: 09/26/2026