Daniela Gasparotto

Aviano, Italy

Last Updated: 09/26/2026

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Found 15 publications

KIT Mutation-NTRK fusion oncogenic driver switch: a novel mechanism of acquired imatinib resistance in GIST.

KIT Mutation-NTRK fusion oncogenic driver switch: a novel mechanism of acquired imatinib resistance in GIST.

Journal: NPJ precision oncology

Published: October 21, 2025

Getting the GIST of long non-coding RNA.

Getting the GIST of long non-coding RNA.

Journal: Biochimica et biophysica acta. Reviews on cancer

Published: October 24, 2024

Whole genome and exome sequencing reference datasets from a multi-center and cross-platform benchmark study.

Whole genome and exome sequencing reference datasets from a multi-center and cross-platform benchmark study.

Journal: Scientific data

Published: March 23, 2021

A Novel Kindred with Familial Gastrointestinal Stromal Tumors Caused by a Rare KIT Germline Mutation (N655K): Clinico-Pathological Presentation and TKI Sensitivity.

A Novel Kindred with Familial Gastrointestinal Stromal Tumors Caused by a Rare KIT Germline Mutation (N655K): Clinico-Pathological Presentation and TKI Sensitivity.

Journal: Journal of personalized medicine

Published: October 16, 2020

Tumor genotype, location, and malignant potential shape the immunogenicity of primary untreated gastrointestinal stromal tumors.

Tumor genotype, location, and malignant potential shape the immunogenicity of primary untreated gastrointestinal stromal tumors.

Journal: JCI insight

Published: July 24, 2020

Neurofibromin C terminus-specific antibody (clone NFC) is a valuable tool for the identification of NF1-inactivated GISTs.

Neurofibromin C terminus-specific antibody (clone NFC) is a valuable tool for the identification of NF1-inactivated GISTs.

Journal: Modern pathology : an official journal of the United States and Canadian Academy of Pathology, Inc

Published: April 19, 2017

Quadruple-Negative GIST Is a Sentinel for Unrecognized Neurofibromatosis Type 1 Syndrome.

Quadruple-Negative GIST Is a Sentinel for Unrecognized Neurofibromatosis Type 1 Syndrome.

Journal: Clinical cancer research : an official journal of the American Association for Cancer Research

Published: January 19, 2016

Concomitant KIT/BRAF and PDGFRA/BRAF mutations are rare events in gastrointestinal stromal tumors.

Concomitant KIT/BRAF and PDGFRA/BRAF mutations are rare events in gastrointestinal stromal tumors.

Journal: Oncotarget

Published: December 30, 2015

Transcriptome sequencing identifies ETV6-NTRK3 as a gene fusion involved in GIST.

Transcriptome sequencing identifies ETV6-NTRK3 as a gene fusion involved in GIST.

Journal: The Journal of pathology

Published: August 31, 2015

Morphologic shift associated with aberrant cytokeratin expression in a GIST patient after tyrosine kinase inhibitors therapy. A case report with a brief review of the literature.

Morphologic shift associated with aberrant cytokeratin expression in a GIST patient after tyrosine kinase inhibitors therapy. A case report with a brief review of the literature.

Journal: Pathology, research and practice

Published: June 17, 2015

KIT, PDGFRA, and BRAF mutational spectrum impacts on the natural history of imatinib-naive localized GIST: a population-based study.

KIT, PDGFRA, and BRAF mutational spectrum impacts on the natural history of imatinib-naive localized GIST: a population-based study.

Journal: The American journal of surgical pathology

Published: May 14, 2015

Imatinib-Sensitizing KIT Mutation in a Carney-Stratakis-Associated GI Stromal Tumor.

Imatinib-Sensitizing KIT Mutation in a Carney-Stratakis-Associated GI Stromal Tumor.

Journal: Journal of clinical oncology : official journal of the American Society of Clinical Oncology

Published: December 31, 2014

Showing 1-12 of 15

Last Updated: 09/26/2026