Danxia Zhou

The State Key Laboratory Of Pharmaceutical Biotechnology And MOE Key Laboratory Of Model Animal For Disease Study
Nanjing, 210000, China

Last Updated: 09/26/2026

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Found 19 publications

HUWE1 in Skeletal Muscle Prevents Muscle Fatigue via Maintaining Iron and Calcium Homeostasis.

HUWE1 in Skeletal Muscle Prevents Muscle Fatigue via Maintaining Iron and Calcium Homeostasis.

Journal: Advanced science (Weinheim, Baden-Wurttemberg, Germany)

Published: August 30, 2025

BRD4 directs myofiber identity and metabolic adaptation through CHD4 cooperation.

BRD4 directs myofiber identity and metabolic adaptation through CHD4 cooperation.

Journal: Nature communications

Published: June 18, 2025

Glucose-Responsive PAGR1-Regulated Skeletal Muscle Gene Program Controls Systemic Glucose Homeostasis and Hepatic Metabolism.

Glucose-Responsive PAGR1-Regulated Skeletal Muscle Gene Program Controls Systemic Glucose Homeostasis and Hepatic Metabolism.

Journal: Advanced science (Weinheim, Baden-Wurttemberg, Germany)

Published: February 13, 2025

Enhancer regulator MLL4 controls skeletal muscle metabolic efficiency by limiting AMPK-mediated fuel catabolism.

Enhancer regulator MLL4 controls skeletal muscle metabolic efficiency by limiting AMPK-mediated fuel catabolism.

Journal: Nature communications

Published: December 04, 2024

Muscle-bone cross-talk through the FNIP1-TFEB-IGF2 axis is associated with bone metabolism in human and mouse.

Muscle-bone cross-talk through the FNIP1-TFEB-IGF2 axis is associated with bone metabolism in human and mouse.

Journal: Science translational medicine

Published: June 05, 2024

Imbalanced Skeletal Muscle Mitochondrial Proteostasis Causes Bone Loss.

Imbalanced Skeletal Muscle Mitochondrial Proteostasis Causes Bone Loss.

Journal: Research (Washington, D.C.)

Published: April 21, 2024

Metabolic Profiling of Cochlear Organoids Identifies α-Ketoglutarate and NAD+ as Limiting Factors for Hair Cell Reprogramming.

Metabolic Profiling of Cochlear Organoids Identifies α-Ketoglutarate and NAD+ as Limiting Factors for Hair Cell Reprogramming.

Journal: Advanced science (Weinheim, Baden-Wurttemberg, Germany)

Published: October 24, 2023

FNIP1 abrogation promotes functional revascularization of ischemic skeletal muscle by driving macrophage recruitment.

FNIP1 abrogation promotes functional revascularization of ischemic skeletal muscle by driving macrophage recruitment.

Journal: Nature communications

Published: February 07, 2023

Mitochondrial proteostasis stress in muscle drives a long-range protective response to alleviate dietary obesity independently of ATF4.

Mitochondrial proteostasis stress in muscle drives a long-range protective response to alleviate dietary obesity independently of ATF4.

Journal: Science advances

Published: July 27, 2022

Proteolytic rewiring of mitochondria by LONP1 directs cell identity switching of adipocytes.

Proteolytic rewiring of mitochondria by LONP1 directs cell identity switching of adipocytes.

Journal: Nature cell biology

Published: June 26, 2022

FNIP1 regulates adipocyte browning and systemic glucose homeostasis in mice by shaping intracellular calcium dynamics.

FNIP1 regulates adipocyte browning and systemic glucose homeostasis in mice by shaping intracellular calcium dynamics.

Journal: The Journal of experimental medicine

Published: December 14, 2021

Disuse-associated loss of the protease LONP1 in muscle impairs mitochondrial function and causes reduced skeletal muscle mass and strength.

Disuse-associated loss of the protease LONP1 in muscle impairs mitochondrial function and causes reduced skeletal muscle mass and strength.

Journal: Nature communications

Published: April 14, 2021

Showing 1-12 of 19

Last Updated: 09/26/2026