Daoxin Q. Wang
Chongqing, China
Last Updated: 09/26/2026
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Found 75 publications
TARM1 may contribute to acute respiratory distress syndrome progression via regulating M1 macrophage polarization.
Journal: Biochemical and biophysical research communications
Published: July 10, 2026
Rab32-mediated macrophage apoptosis and apoptotic body release promote M1 polarization in ARDS via the Cxcl11/Ccl4/NF-κB pathway.
Journal: International immunopharmacology
Published: January 09, 2026
Clusterin ameliorates LPS-induced ARDS by inhibiting the Wnt/β-catenin pathway.
Journal: Clinical science (London, England : 1979)
Published: December 30, 2025
Albumin-corrected anion gap as a predictor of 28-day mortality in acute respiratory distress syndrome: A machine learning-based retrospective study.
Journal: PloS one
Published: July 04, 2025
Inhibition of antiapoptotic BCL-2 proteins with Lipo-NPs induces fibroblast apoptosis, alleviating pulmonary fibrosis in VILI mice.
Journal: Respiratory research
Published: June 29, 2025
Association Between the Non-High-Density Lipoprotein Cholesterol-to-High-Density Lipoprotein Cholesterol Ratio (NHHR) and Mortality in Patients with COPD: Evidence From the NHANES 1999-2018.
Journal: International journal of chronic obstructive pulmonary disease
Published: November 25, 2024
CGRP alleviates lipopolysaccharide-induced ARDS inflammation via the HIF-1α signaling pathway.
Journal: Clinical science (London, England : 1979)
Published: November 20, 2024
Celastrol ameliorates lipopolysaccharide (LPS)-induced acute lung injury by improving mitochondrial function through AMPK/PGC-1α/Nrf1-dependent mechanism.
Journal: Free radical biology & medicine
Published: September 23, 2024
MFGE8 regulates the EndoMT of HLMECs through the BMP signaling pathway and fibrosis in acute lung injury.
Journal: Respiratory research
Published: September 14, 2024
Omentin-1 ameliorates pulmonary arterial hypertension by inhibiting endoplasmic reticulum stress through AMPKα signaling.
Journal: Clinical and experimental hypertension (New York, N.Y. : 1993)
Published: March 25, 2024
S1PR3 inhibition protects against LPS-induced ARDS by inhibiting NF-κB and improving mitochondrial oxidative phosphorylation.
Journal: Journal of translational medicine
Published: January 24, 2024
Last Updated: 09/26/2026