Dasan M. Cibi
Singapore, Singapore
Last Updated: 09/26/2026
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Found 11 publications
YAP/TAZ are crucial regulators of macrophage-mediated pulmonary inflammation and fibrosis after bleomycin-induced injury.
Journal: The European respiratory journal
Published: September 12, 2023
Loss of Yap/Taz in cardiac fibroblasts attenuates adverse remodelling and improves cardiac function.
Loss of Yap/Taz in cardiac fibroblasts attenuates adverse remodelling and improves cardiac function.
Journal: Cardiovascular research
Published: February 16, 2021
YAP/TAZ deficiency reprograms macrophage phenotype and improves infarct healing and cardiac function after myocardial infarction.
Journal: PLoS biology
Published: October 18, 2019
Prdm16 Deficiency Leads to Age-Dependent Cardiac Hypertrophy, Adverse Remodeling, Mitochondrial Dysfunction, and Heart Failure.
Journal: Cell reports
Published: September 24, 2019
Deficiency in the secreted protein Semaphorin3d causes abnormal parathyroid development in mice.
Journal: The Journal of biological chemistry
Published: December 07, 2018
Semaphorin 3E/PlexinD1 signaling is required for cardiac ventricular compaction.
Journal: JCI insight
Published: October 30, 2018
Epicardial YAP/TAZ orchestrate an immunosuppressive response following myocardial infarction.
Journal: The Journal of clinical investigation
Published: May 25, 2016
In Vitro Culture of Epicardial Cells From Mouse Embryonic Heart.
Journal: Journal of visualized experiments : JoVE
Published: May 12, 2016
Showing 1-11 of 11
Last Updated: 09/26/2026