David H. Jang

Emergency Medicine

3400 Spruce St
Philadelphia, PA 19104

Last Updated: 09/26/2026

Save publications for later

Sign Up

Not sure about your diagnosis?

Check Your Symptoms

Found 60 publications

Mitochondrial Dysfunction, Cerebral Metabolic Crisis, and Exploratory Bioenergetic Biomarkers in a Translational Swine Model of Acute Carbon Monoxide Poisoning.

Mitochondrial Dysfunction, Cerebral Metabolic Crisis, and Exploratory Bioenergetic Biomarkers in a Translational Swine Model of Acute Carbon Monoxide Poisoning.

Journal: Toxicological sciences : an official journal of the Society of Toxicology

Published: July 30, 2026

Targeting Mitochondrial Dysfunction in Acute Organophosphate Toxicity: A Preliminary Evaluation of Cyclosporine A.

Targeting Mitochondrial Dysfunction in Acute Organophosphate Toxicity: A Preliminary Evaluation of Cyclosporine A.

Journal: Journal of medical toxicology : official journal of the American College of Medical Toxicology

Published: January 28, 2026

Dose-dependent cerebral metabolic impairment in a swine model of carbon monoxide poisoning.

Dose-dependent cerebral metabolic impairment in a swine model of carbon monoxide poisoning.

Journal: Neurotoxicology

Published: December 17, 2025

Plasma and Imaging Biomarker Changes Following Rotational and Contusional Models of Traumatic Brain Injury in Adolescent Pigs.

Plasma and Imaging Biomarker Changes Following Rotational and Contusional Models of Traumatic Brain Injury in Adolescent Pigs.

Journal: Journal of neurotrauma

Published: December 10, 2025

Timing of inotropic support is associated with mortality in patients with acute decompensated heart failure-associated cardiogenic shock.

Timing of inotropic support is associated with mortality in patients with acute decompensated heart failure-associated cardiogenic shock.

Journal: Intensive care medicine experimental

Published: June 02, 2025

Attenuation of mitochondrial dysfunction in a ventricular fibrillation swine model of cardiac arrest treated with carbon monoxide.

Attenuation of mitochondrial dysfunction in a ventricular fibrillation swine model of cardiac arrest treated with carbon monoxide.

Journal: Resuscitation

Published: March 05, 2025

The authors reply.

The authors reply.

Journal: Critical care medicine

Published: January 08, 2025

Blood Cells as a Cellular Biomarker for Mitochondrial Function in a Experimental Model of Acute Carbon Monoxide Poisoning with Treatment.

Blood Cells as a Cellular Biomarker for Mitochondrial Function in a Experimental Model of Acute Carbon Monoxide Poisoning with Treatment.

Journal: Journal of medical toxicology : official journal of the American College of Medical Toxicology

Published: December 25, 2024

The application of cell-free DNA methylation patterns in critical illnesses: A protocol paper.

The application of cell-free DNA methylation patterns in critical illnesses: A protocol paper.

Journal: PloS one

Published: September 26, 2024

American College of Medical Toxicology Research Agenda 2024-2030.

American College of Medical Toxicology Research Agenda 2024-2030.

Journal: Journal of medical toxicology : official journal of the American College of Medical Toxicology

Published: May 22, 2024

Anaerobic Lactate Production Is Associated With Decreased Microcirculatory Blood Flow and Decreased Mitochondrial Respiration Following Cardiovascular Surgery With Cardiopulmonary Bypass.

Anaerobic Lactate Production Is Associated With Decreased Microcirculatory Blood Flow and Decreased Mitochondrial Respiration Following Cardiovascular Surgery With Cardiopulmonary Bypass.

Journal: Critical care medicine

Published: April 05, 2024

Carbon monoxide as a cellular protective agent in a swine model of cardiac arrest protocol.

Carbon monoxide as a cellular protective agent in a swine model of cardiac arrest protocol.

Journal: PloS one

Published: August 03, 2023

Showing 1-12 of 60

Last Updated: 09/26/2026