Delphine Goehrig

Lyon, France

Last Updated: 09/26/2026

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Found 18 publications

The Proinflammatory Secretome of Senescent Cells Can Be Controlled by a HIF2A-Dependent Upregulation and a FURIN-Dependent Cleavage of the ANGPTL4 Secreted Factor.

The Proinflammatory Secretome of Senescent Cells Can Be Controlled by a HIF2A-Dependent Upregulation and a FURIN-Dependent Cleavage of the ANGPTL4 Secreted Factor.

Journal: Aging cell
Published: January 22, 2025

DNA methylation protects cancer cells against senescence.

DNA methylation protects cancer cells against senescence.

Journal: Nature communications
Published: September 27, 2024

A non-canonical role of ELN protects from cellular senescence by limiting iron-dependent regulation of gene expression.

A non-canonical role of ELN protects from cellular senescence by limiting iron-dependent regulation of gene expression.

Journal: Redox biology
Published: February 23, 2024

Targeting chemoresistant senescent pancreatic cancer cells improves conventional treatment efficacy.

Targeting chemoresistant senescent pancreatic cancer cells improves conventional treatment efficacy.

Journal: Molecular biomedicine
Published: September 08, 2022

Loss of Pla2r1 decreases cellular senescence and age-related alterations caused by aging and Western diets.

Loss of Pla2r1 decreases cellular senescence and age-related alterations caused by aging and Western diets.

Journal: Aging cell
Published: August 02, 2022

Transformed cells after senescence give rise to more severe tumor phenotypes than transformed non-senescent cells.

Transformed cells after senescence give rise to more severe tumor phenotypes than transformed non-senescent cells.

Journal: Cancer letters
Published: May 30, 2022

NF-κB-dependent secretome of senescent cells can trigger neuroendocrine transdifferentiation of breast cancer cells.

NF-κB-dependent secretome of senescent cells can trigger neuroendocrine transdifferentiation of breast cancer cells.

Journal: Aging cell
Published: July 21, 2021

PLA2R1 promotes DNA damage and inhibits spontaneous tumor formation during aging.

PLA2R1 promotes DNA damage and inhibits spontaneous tumor formation during aging.

Journal: Cell death & disease
Published: March 24, 2020

Phospholipase A2 receptor 1 promotes lung cell senescence and emphysema in obstructive lung disease.

Phospholipase A2 receptor 1 promotes lung cell senescence and emphysema in obstructive lung disease.

Journal: The European respiratory journal
Published: March 18, 2020

Oncogene-Induced Senescence Limits the Progression of Pancreatic Neoplasia through Production of Activin A.

Oncogene-Induced Senescence Limits the Progression of Pancreatic Neoplasia through Production of Activin A.

Journal: Cancer research
Published: December 19, 2019

Generation of a conditional transgenic mouse model expressing human Phospholipase A2 Receptor 1.

Generation of a conditional transgenic mouse model expressing human Phospholipase A2 Receptor 1.

Journal: Scientific reports
Published: September 26, 2019

Calcium channel ITPR2 and mitochondria-ER contacts promote cellular senescence and aging.

Calcium channel ITPR2 and mitochondria-ER contacts promote cellular senescence and aging.

Journal: Nature communications
Published: August 26, 2019
Showing 1-12 of 18

Last Updated: 09/26/2026