Denisse M. Valladares

1509 Pedro De Valdivia Av, Providencia
Santiago, RM, Chile

Last Updated: 09/26/2026

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Found 9 publications

Changes in oxidative stress, inflammation and muscle damage markers following eccentric versus concentric cycling in older adults.

Changes in oxidative stress, inflammation and muscle damage markers following eccentric versus concentric cycling in older adults.

Journal: European journal of applied physiology

Published: June 19, 2019

IP3 receptor blockade restores autophagy and mitochondrial function in skeletal muscle fibers of dystrophic mice.

IP3 receptor blockade restores autophagy and mitochondrial function in skeletal muscle fibers of dystrophic mice.

Journal: Biochimica et biophysica acta. Molecular basis of disease

Published: May 05, 2018

Mitochondrial Calcium Increase Induced by RyR1 and IP3R Channel Activation After Membrane Depolarization Regulates Skeletal Muscle Metabolism.

Mitochondrial Calcium Increase Induced by RyR1 and IP3R Channel Activation After Membrane Depolarization Regulates Skeletal Muscle Metabolism.

Journal: Frontiers in physiology

Published: January 25, 2018

GSK-3β/NFAT Signaling Is Involved in Testosterone-Induced Cardiac Myocyte Hypertrophy.

GSK-3β/NFAT Signaling Is Involved in Testosterone-Induced Cardiac Myocyte Hypertrophy.

Journal: PloS one

Published: August 24, 2016

NOX2 Inhibition Impairs Early Muscle Gene Expression Induced by a Single Exercise Bout.

NOX2 Inhibition Impairs Early Muscle Gene Expression Induced by a Single Exercise Bout.

Journal: Frontiers in physiology

Published: May 04, 2016

Characterization of a multiprotein complex involved in excitation-transcription coupling of skeletal muscle.

Characterization of a multiprotein complex involved in excitation-transcription coupling of skeletal muscle.

Journal: Skeletal muscle

Published: January 19, 2016

Altered ROS production, NF-κB activation and interleukin-6 gene expression induced by electrical stimulation in dystrophic mdx skeletal muscle cells.

Altered ROS production, NF-κB activation and interleukin-6 gene expression induced by electrical stimulation in dystrophic mdx skeletal muscle cells.

Journal: Biochimica et biophysica acta

Published: December 03, 2014

Nifedipine treatment reduces resting calcium concentration, oxidative and apoptotic gene expression, and improves muscle function in dystrophic mdx mice.

Nifedipine treatment reduces resting calcium concentration, oxidative and apoptotic gene expression, and improves muscle function in dystrophic mdx mice.

Journal: PloS one

Published: June 26, 2013

Electrical stimuli are anti-apoptotic in skeletal muscle via extracellular ATP. Alteration of this signal in Mdx mice is a likely cause of dystrophy.

Electrical stimuli are anti-apoptotic in skeletal muscle via extracellular ATP. Alteration of this signal in Mdx mice is a likely cause of dystrophy.

Journal: PloS one

Published: May 17, 2013

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Last Updated: 09/26/2026