Dhiviya Vedagiri

Uppal Road
Hyderabad, TG 500007, India

Last Updated: 09/26/2026

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Found 11 publications

A glaucoma-associated OPTN polymorphism, M98K sensitizes retinal cells to endoplasmic reticulum stress and tumour necrosis factor α.

A glaucoma-associated OPTN polymorphism, M98K sensitizes retinal cells to endoplasmic reticulum stress and tumour necrosis factor α.

Journal: The FEBS journal

Published: August 08, 2022

Correction for Vedagiri et al., "Retinoic Acid-Inducible Gene I-Like Receptors Activate Snail To Limit RNA Viral Infections".

Correction for Vedagiri et al., "Retinoic Acid-Inducible Gene I-Like Receptors Activate Snail To Limit RNA Viral Infections".

Journal: Journal of virology

Published: January 26, 2022

Glycolytic inhibitor 2-deoxy-d-glucose attenuates SARS-CoV-2 multiplication in host cells and weakens the infective potential of progeny virions.

Glycolytic inhibitor 2-deoxy-d-glucose attenuates SARS-CoV-2 multiplication in host cells and weakens the infective potential of progeny virions.

Journal: Life sciences

Published: December 07, 2021

Equine immunoglobulin fragment F(ab')2 displays high neutralizing capability against multiple SARS-CoV-2 variants.

Equine immunoglobulin fragment F(ab')2 displays high neutralizing capability against multiple SARS-CoV-2 variants.

Journal: Clinical immunology (Orlando, Fla.)

Published: November 08, 2021

Retinoic Acid-Inducible Gene I-Like Receptors Activate Snail To Limit RNA Viral Infections.

Retinoic Acid-Inducible Gene I-Like Receptors Activate Snail To Limit RNA Viral Infections.

Journal: Journal of virology

Published: August 11, 2021

Inactivation of SARS-CoV-2 by β-propiolactone causes aggregation of viral particles and loss of antigenic potential.

Inactivation of SARS-CoV-2 by β-propiolactone causes aggregation of viral particles and loss of antigenic potential.

Journal: Virus research

Published: May 14, 2021

Easing diagnosis and pushing the detection limits of SARS-CoV-2.

Easing diagnosis and pushing the detection limits of SARS-CoV-2.

Journal: Biology methods & protocols

Published: July 19, 2020

mTORC1 restricts hepatitis C virus RNA replication through ULK1-mediated suppression of miR-122 and facilitates post-replication events.

mTORC1 restricts hepatitis C virus RNA replication through ULK1-mediated suppression of miR-122 and facilitates post-replication events.

Journal: The Journal of general virology

Published: December 11, 2019

Unconventional MAPK-GSK-3β Pathway Behind Atypical Epithelial-Mesenchymal Transition In Hepatocellular Carcinoma.

Unconventional MAPK-GSK-3β Pathway Behind Atypical Epithelial-Mesenchymal Transition In Hepatocellular Carcinoma.

Journal: Scientific reports

Published: May 15, 2017

An Atypical System for Studying Epithelial-Mesenchymal Transition in Hepatocellular Carcinoma.

An Atypical System for Studying Epithelial-Mesenchymal Transition in Hepatocellular Carcinoma.

Journal: Scientific reports

Published: October 29, 2015

A unique phosphorylation-dependent eIF4E assembly on 40S ribosomes co-ordinated by hepatitis C virus protein NS5A that activates internal ribosome entry site translation.

A unique phosphorylation-dependent eIF4E assembly on 40S ribosomes co-ordinated by hepatitis C virus protein NS5A that activates internal ribosome entry site translation.

Journal: The Biochemical journal

Published: June 05, 2014

Showing 1-11 of 11

Last Updated: 09/26/2026