Diana Teen-Bou

Universitat Autonoma De Barcelona And Centro De Investigación Biomédica En Red CV, CIBER CV
Madrid, MD, Spain

Last Updated: 09/26/2026

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Found 9 publications

Glycative Stress Disrupts the Mitochondrial-Lysosome Axis and Promotes Geroconversion in Aging Cardiomyocytes.

Glycative Stress Disrupts the Mitochondrial-Lysosome Axis and Promotes Geroconversion in Aging Cardiomyocytes.

Journal: Aging cell

Published: June 20, 2025

Dicarbonyl stress and mitochondrial dysfunction in the aged heart.

Dicarbonyl stress and mitochondrial dysfunction in the aged heart.

Journal: Aging

Published: January 24, 2023

Defective dimerization of FoF1-ATP synthase secondary to glycation favors mitochondrial energy deficiency in cardiomyocytes during aging.

Defective dimerization of FoF1-ATP synthase secondary to glycation favors mitochondrial energy deficiency in cardiomyocytes during aging.

Journal: Aging cell

Published: October 28, 2021

Mitochondrial ROS and mitochondria-targeted antioxidants in the aged heart.

Mitochondrial ROS and mitochondria-targeted antioxidants in the aged heart.

Journal: Free radical biology & medicine

Published: December 04, 2020

Enhancing Glycolysis Protects against Ischemia-Reperfusion Injury by Reducing ROS Production.

Enhancing Glycolysis Protects against Ischemia-Reperfusion Injury by Reducing ROS Production.

Journal: Metabolites

Published: March 19, 2020

Cardiomyocyte ageing and cardioprotection: consensus document from the ESC working groups cell biology of the heart and myocardial function.

Cardiomyocyte ageing and cardioprotection: consensus document from the ESC working groups cell biology of the heart and myocardial function.

Journal: Cardiovascular research

Published: January 28, 2020

Cyclophilins A and B oppositely regulate renal tubular epithelial cell phenotype.

Cyclophilins A and B oppositely regulate renal tubular epithelial cell phenotype.

Journal: Journal of molecular cell biology

Published: January 28, 2019

Ryanodine Receptor Glycation Favors Mitochondrial Damage in the Senescent Heart.

Ryanodine Receptor Glycation Favors Mitochondrial Damage in the Senescent Heart.

Journal: Circulation

Published: December 28, 2018

Evolution and bad prognostic value of advanced glycation end products after acute heart failure: relation with body composition.

Evolution and bad prognostic value of advanced glycation end products after acute heart failure: relation with body composition.

Journal: Cardiovascular diabetology

Published: June 30, 2017

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Last Updated: 09/26/2026