Diane J. Bakker
Amsterdam, NH 1105AZ, Netherlands
Last Updated: 09/26/2026
Save publications for later
Sign Up
Not sure about your diagnosis?
Check Your Symptoms
Found 15 publications
Metabolic dependency of malonate protection against cardiac ischemia-reperfusion injury.
Journal: Basic research in cardiology
Published: November 24, 2025
The early metabolic cardiac targets of empagliflozin during the development of heart failure, independent of SGLT2 inhibition.
Journal: Metabolism: clinical and experimental
Published: September 19, 2025
Sodium-glucose co-transporters (SGLT2) inhibitors prevent lipid droplets formation in vascular inflammation or lipid overload by SGLT2-independent mechanism.
Journal: Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie
Published: December 23, 2024
Empagliflozin prevents heart failure through inhibition of the NHE1-NO pathway, independent of SGLT2.
Journal: Basic research in cardiology
Published: March 05, 2024
Cardioprotecive Properties of Known Agents in Rat Ischemia-Reperfusion Model Under Clinically Relevant Conditions: Only the NAD Precursor Nicotinamide Riboside Reduces Infarct Size in Presence of Fentanyl, Midazolam and Cangrelor, but Not Propofol.
Journal: Frontiers in cardiovascular medicine
Published: May 20, 2021
The Redox Modulating Sonlicromanol Active Metabolite KH176m and the Antioxidant MPG Protect Against Short-Duration Cardiac Ischemia-Reperfusion Injury.
Journal: Cardiovascular drugs and therapy
Published: April 12, 2021
Empagliflozin Decreases Lactate Generation in an NHE-1 Dependent Fashion and Increases α-Ketoglutarate Synthesis From Palmitate in Type II Diabetic Mouse Hearts.
Journal: Frontiers in cardiovascular medicine
Published: August 06, 2020
NLRX1 Deletion Increases Ischemia-Reperfusion Damage and Activates Glucose Metabolism in Mouse Heart.
Journal: Frontiers in immunology
Published: August 05, 2020
Cardiomyocyte Progenitor Cells as a Functional Gene Delivery Vehicle for Long-Term Biological Pacing.
Journal: Molecules (Basel, Switzerland)
Published: November 28, 2018
Genetic variation in GNB5 causes bradycardia by augmenting the cholinergic response via increased acetylcholine-activated potassium current (IK,ACh).
Journal: Disease models & mechanisms
Published: November 08, 2018
Last Updated: 09/26/2026