Dong G. Lee

Daegu, Republic of Korea

Last Updated: 09/26/2026

Save publications for later

Sign Up

Not sure about your diagnosis?

Check Your Symptoms

Found 14 publications

Amyloid beta oligomers-induced parkin aggravates ER stress-mediated cell death through a positive feedback loop.

Amyloid beta oligomers-induced parkin aggravates ER stress-mediated cell death through a positive feedback loop.

Journal: Neurochemistry international

Published: October 13, 2021

Amyloid-beta oligomers induce Parkin-mediated mitophagy by reducing Miro1.

Amyloid-beta oligomers induce Parkin-mediated mitophagy by reducing Miro1.

Journal: The Biochemical journal

Published: June 19, 2020

Isoliquiritigenin Reduces LPS-Induced Inflammation by Preventing Mitochondrial Fission in BV-2 Microglial Cells.

Isoliquiritigenin Reduces LPS-Induced Inflammation by Preventing Mitochondrial Fission in BV-2 Microglial Cells.

Journal: Inflammation

Published: April 17, 2020

Peroxiredoxin 5 deficiency exacerbates iron overload-induced neuronal death via ER-mediated mitochondrial fission in mouse hippocampus.

Peroxiredoxin 5 deficiency exacerbates iron overload-induced neuronal death via ER-mediated mitochondrial fission in mouse hippocampus.

Journal: Cell death & disease

Published: September 16, 2019

Peroxiredoxin 5 prevents diethylhexyl phthalate-induced neuronal cell death by inhibiting mitochondrial fission in mouse hippocampal HT-22 cells.

Peroxiredoxin 5 prevents diethylhexyl phthalate-induced neuronal cell death by inhibiting mitochondrial fission in mouse hippocampal HT-22 cells.

Journal: Neurotoxicology

Published: March 18, 2019

Peroxiredoxin 4 ameliorates amyloid beta oligomer-mediated apoptosis by inhibiting ER-stress in HT-22 hippocampal neuron cells.

Peroxiredoxin 4 ameliorates amyloid beta oligomer-mediated apoptosis by inhibiting ER-stress in HT-22 hippocampal neuron cells.

Journal: Cell biology and toxicology

Published: January 08, 2019

Peroxiredoxin I maintains luteal function by regulating unfolded protein response.

Peroxiredoxin I maintains luteal function by regulating unfolded protein response.

Journal: Reproductive biology and endocrinology : RB&E

Published: April 26, 2018

Peroxiredoxin 5 prevents iron overload-induced neuronal death by inhibiting mitochondrial fragmentation and endoplasmic reticulum stress in mouse hippocampal HT-22 cells.

Peroxiredoxin 5 prevents iron overload-induced neuronal death by inhibiting mitochondrial fragmentation and endoplasmic reticulum stress in mouse hippocampal HT-22 cells.

Journal: The international journal of biochemistry & cell biology

Published: April 06, 2018

Peroxiredoxin 2 deficiency accelerates age-related ovarian failure through the reactive oxygen species-mediated JNK pathway in mice.

Peroxiredoxin 2 deficiency accelerates age-related ovarian failure through the reactive oxygen species-mediated JNK pathway in mice.

Journal: Free radical biology & medicine

Published: February 14, 2018

Isoliquiritigenin attenuates glutamate-induced mitochondrial fission via calcineurin-mediated Drp1 dephosphorylation in HT22 hippocampal neuron cells.

Isoliquiritigenin attenuates glutamate-induced mitochondrial fission via calcineurin-mediated Drp1 dephosphorylation in HT22 hippocampal neuron cells.

Journal: Neurotoxicology

Published: December 29, 2017

Peroxiredoxin 5 Decreases Beta-Amyloid-Mediated Cyclin-Dependent Kinase 5 Activation Through Regulation of Ca2+-Mediated Calpain Activation.

Peroxiredoxin 5 Decreases Beta-Amyloid-Mediated Cyclin-Dependent Kinase 5 Activation Through Regulation of Ca2+-Mediated Calpain Activation.

Journal: Antioxidants & redox signaling

Published: March 31, 2017

Iron overload-induced calcium signals modulate mitochondrial fragmentation in HT-22 hippocampal neuron cells.

Iron overload-induced calcium signals modulate mitochondrial fragmentation in HT-22 hippocampal neuron cells.

Journal: Toxicology

Published: May 12, 2016

Showing 1-12 of 14

Last Updated: 09/26/2026