Dongwei W. Liu

Zhengzhou, China

Last Updated: 09/26/2026

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Found 99 publications

WTAP Deficiency Promotes Podocyte PANoptosis and FSGS via SPP1 Signaling.

WTAP Deficiency Promotes Podocyte PANoptosis and FSGS via SPP1 Signaling.

Journal: Circulation research

Published: July 07, 2026

WTAP Deficiency Promotes Podocyte PANoptosis and FSGS via SPP1 Signaling.

WTAP Deficiency Promotes Podocyte PANoptosis and FSGS via SPP1 Signaling.

Journal: Circulation research

Published: July 07, 2026

Development and preliminary clinical application of a time-resolved fluoroimmunoassay for anti-rituximab antibodies in membranous nephropathy.

Development and preliminary clinical application of a time-resolved fluoroimmunoassay for anti-rituximab antibodies in membranous nephropathy.

Journal: Bioanalysis

Published: June 25, 2026

USP30-Mediated Deubiquitination of PEX5 Suppresses Pexophagy to Drive Tubular Injury in Diabetic Kidney Disease.

USP30-Mediated Deubiquitination of PEX5 Suppresses Pexophagy to Drive Tubular Injury in Diabetic Kidney Disease.

Journal: Advanced science (Weinheim, Baden-Wurttemberg, Germany)

Published: April 24, 2026

Epigenetic Regulation of Ferroptosis in Chronic Kidney Disease: Mechanisms and Implications.

Epigenetic Regulation of Ferroptosis in Chronic Kidney Disease: Mechanisms and Implications.

Journal: Research (Washington, D.C.)

Published: June 30, 2025

Benzo[b]fluoranthene is involved in idiopathic membranous nephropathy by inducing podocyte injury.

Benzo[b]fluoranthene is involved in idiopathic membranous nephropathy by inducing podocyte injury.

Journal: Journal of translational internal medicine

Published: May 30, 2025

Histone methyltransferase EZH2 drives podocyte injury and senescence in diabetic nephropathy through STAT3 activation.

Histone methyltransferase EZH2 drives podocyte injury and senescence in diabetic nephropathy through STAT3 activation.

Journal: Cellular and molecular life sciences : CMLS

Published: April 23, 2025

MYO1C is a urinary extracellular vesicle biomarker and mediator of podocyte injury in diabetic nephropathy.

MYO1C is a urinary extracellular vesicle biomarker and mediator of podocyte injury in diabetic nephropathy.

Journal: JCI insight

Published: April 16, 2025

The mechanism of β-hydroxybutyrylation in G6PDX regulated by β-hydroxybutyric acid on alleviating podocyte injury induced by high glucose

The mechanism of β-hydroxybutyrylation in G6PDX regulated by β-hydroxybutyric acid on alleviating podocyte injury induced by high glucose

Journal: Zhonghua yi xue za zhi

Published: March 20, 2025

Progress and prospects of finerenone in the treatment of type 2 diabetes mellitus related chronic kidney disease

Progress and prospects of finerenone in the treatment of type 2 diabetes mellitus related chronic kidney disease

Journal: Zhonghua yi xue za zhi

Published: March 20, 2025

Bone marrow mesenchymal stem cells-derived exosomes protect against β-cell destruction models and kidney injury by suppressing ferroptosis.

Bone marrow mesenchymal stem cells-derived exosomes protect against β-cell destruction models and kidney injury by suppressing ferroptosis.

Journal: Scientific reports

Published: February 10, 2025

Lactate and Lactylation in AKI-to-CKD: Epigenetic Regulation and Therapeutic Opportunities.

Lactate and Lactylation in AKI-to-CKD: Epigenetic Regulation and Therapeutic Opportunities.

Journal: Cell proliferation

Published: December 27, 2024

Showing 1-12 of 99

Last Updated: 09/26/2026