Dongzhi L. Wang
301 Yanchang Road
Shanghai, 200072, China
Shanghai, 200072, China
Last Updated: 09/26/2026
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Found 18 publications
Systolic pressure overload caused pulmonary oxidative stress, vessel remodeling and severe microvascular thrombosis in CD40 knockout mice through promoting platelet aggregation.
Journal: Redox biology
Published: January 05, 2026
Genetic inhibition of IL-12β suppresses systolic overload-induced cardiac oxidative stress, inflammation, and heart failure development.
Journal: Redox biology
Published: December 02, 2025
Genetic inhibition of IL-12β suppresses systolic overload-induced cardiac inflammation and heart failure development.
Journal: bioRxiv : the preprint server for biology
Published: September 18, 2025
PHD2 Deletion in CD8+ T Cells Worsens TAC-Induced Cardiac Inflammation, Heart Failure, and Pulmonary Remodeling.
Journal: Hypertension (Dallas, Tex. : 1979)
Published: September 17, 2025
Pharmacological inhibition of IL12β is effective in treating pressure overload-induced cardiac inflammation and heart failure.
Journal: Frontiers in immunology
Published: May 08, 2025
CD8 T cells promote heart failure progression in mice with preexisting left ventricular dysfunction.
CD8 T cells promote heart failure progression in mice with preexisting left ventricular dysfunction.
Journal: Frontiers in immunology
Published: July 28, 2024
Role of miRNA-499-5p in patients with atrial fibrillation and heart failure.
Journal: Journal of biological regulators and homeostatic agents
Published: July 23, 2020
Systolic overload-induced pulmonary inflammation, fibrosis, oxidative stress and heart failure progression through interleukin-1β.
Journal: Journal of molecular and cellular cardiology
Published: January 16, 2020
Dimethylarginine dimethylaminohydrolase 1 deficiency aggravates monocrotaline-induced pulmonary oxidative stress, pulmonary arterial hypertension and right heart failure in rats.
Journal: International journal of cardiology
Published: April 25, 2019
Isolevuglandin scavenger attenuates pressure overload-induced cardiac oxidative stress, cardiac hypertrophy, heart failure and lung remodeling.
Journal: Free radical biology & medicine
Published: April 20, 2019
Short term Pm2.5 exposure caused a robust lung inflammation, vascular remodeling, and exacerbated transition from left ventricular failure to right ventricular hypertrophy.
Journal: Redox biology
Published: December 22, 2018
Last Updated: 09/26/2026