Doris Dea

Verdun, QC H4H1R3, Canada

Last Updated: 09/26/2026

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Found 11 publications

TLR4 Gene Expression and Pro-Inflammatory Cytokines in Alzheimer's Disease and in Response to Hippocampal Deafferentation in Rodents.

TLR4 Gene Expression and Pro-Inflammatory Cytokines in Alzheimer's Disease and in Response to Hippocampal Deafferentation in Rodents.

Journal: Journal of Alzheimer's disease : JAD

Published: May 22, 2018

Loss-of-Function PCSK9 Mutations Are Not Associated With Alzheimer Disease.

Loss-of-Function PCSK9 Mutations Are Not Associated With Alzheimer Disease.

Journal: Journal of geriatric psychiatry and neurology

Published: March 23, 2018

CDK5RAP2 gene and tau pathophysiology in late-onset sporadic Alzheimer's disease.

CDK5RAP2 gene and tau pathophysiology in late-onset sporadic Alzheimer's disease.

Journal: Alzheimer's & dementia : the journal of the Alzheimer's Association

Published: September 22, 2017

Alterations in cholesterol metabolism-related genes in sporadic Alzheimer's disease.

Alterations in cholesterol metabolism-related genes in sporadic Alzheimer's disease.

Journal: Neurobiology of aging

Published: September 21, 2017

Isoprenoids and tau pathology in sporadic Alzheimer's disease.

Isoprenoids and tau pathology in sporadic Alzheimer's disease.

Journal: Neurobiology of aging

Published: September 06, 2017

Profiles of β-Amyloid Peptides and Key Secretases in Brain Autopsy Samples Differ with Sex and APOE ε4 Status: Impact for Risk and Progression of Alzheimer Disease.

Profiles of β-Amyloid Peptides and Key Secretases in Brain Autopsy Samples Differ with Sex and APOE ε4 Status: Impact for Risk and Progression of Alzheimer Disease.

Journal: Neuroscience

Published: August 31, 2017

Multimodal Imaging in Rat Model Recapitulates Alzheimer's Disease Biomarkers Abnormalities.

Multimodal Imaging in Rat Model Recapitulates Alzheimer's Disease Biomarkers Abnormalities.

Journal: The Journal of neuroscience : the official journal of the Society for Neuroscience

Published: May 17, 2017

Butyrylcholinesterase K and Apolipoprotein E-ɛ4 Reduce the Age of Onset of Alzheimer's Disease, Accelerate Cognitive Decline, and Modulate Donepezil Response in Mild Cognitively Impaired Subjects.

Butyrylcholinesterase K and Apolipoprotein E-ɛ4 Reduce the Age of Onset of Alzheimer's Disease, Accelerate Cognitive Decline, and Modulate Donepezil Response in Mild Cognitively Impaired Subjects.

Journal: Journal of Alzheimer's disease : JAD

Published: August 29, 2016

Effects of rs3846662 Variants on HMGCR mRNA and Protein Levels and on Markers of Alzheimer's Disease Pathology.

Effects of rs3846662 Variants on HMGCR mRNA and Protein Levels and on Markers of Alzheimer's Disease Pathology.

Journal: Journal of molecular neuroscience : MN

Published: May 11, 2015

HMGCR is a genetic modifier for risk, age of onset and MCI conversion to Alzheimer's disease in a three cohorts study.

HMGCR is a genetic modifier for risk, age of onset and MCI conversion to Alzheimer's disease in a three cohorts study.

Journal: Molecular psychiatry

Published: February 10, 2014

Apolipoprotein E and lipid homeostasis in the etiology and treatment of sporadic Alzheimer's disease.

Apolipoprotein E and lipid homeostasis in the etiology and treatment of sporadic Alzheimer's disease.

Journal: Neurobiology of aging

Published: November 06, 2013

Showing 1-11 of 11

Last Updated: 09/26/2026