E R. Paalberends-Witjas
Amsterdam, NH, Netherlands
Last Updated: 09/26/2026
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Found 10 publications
The homozygous K280N troponin T mutation alters cross-bridge kinetics and energetics in human HCM.
Journal: The Journal of general physiology
Published: June 21, 2018
Faster cross-bridge detachment and increased tension cost in human hypertrophic cardiomyopathy with the R403Q MYH7 mutation.
Journal: The Journal of physiology
Published: June 15, 2014
Gene-specific increase in the energetic cost of contraction in hypertrophic cardiomyopathy caused by thick filament mutations.
Journal: Cardiovascular research
Published: May 20, 2014
Phosphorylation of protein kinase C sites Ser42/44 decreases Ca(2+)-sensitivity and blunts enhanced length-dependent activation in response to protein kinase A in human cardiomyocytes.
Journal: Archives of biochemistry and biophysics
Published: March 10, 2014
ENerGetIcs in hypertrophic cardiomyopathy: traNslation between MRI, PET and cardiac myofilament function (ENGINE study).
Journal: Netherlands heart journal : monthly journal of the Netherlands Society of Cardiology and the Netherlands Heart Foundation
Published: October 12, 2013
Cardiac myosin-binding protein C: hypertrophic cardiomyopathy mutations and structure-function relationships.
Journal: Pflugers Archiv : European journal of physiology
Published: October 07, 2013
GSK3β phosphorylates newly identified site in the proline-alanine-rich region of cardiac myosin-binding protein C and alters cross-bridge cycling kinetics in human: short communication.
Journal: Circulation research
Published: January 02, 2013
Familial hypertrophic cardiomyopathy: functional effects of myosin mutation R723G in cardiomyocytes.
Familial hypertrophic cardiomyopathy: functional effects of myosin mutation R723G in cardiomyocytes.
Journal: Journal of molecular and cellular cardiology
Published: December 10, 2012
Showing 1-10 of 10
Last Updated: 09/26/2026