Eduardo A. Sagredo
Santiago, RM, Chile
Last Updated: 09/26/2026
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Found 17 publications
ADAR1 Regulates Alternative Splicing Through an RNA Editing-Independent Mechanism.
Journal: International journal of molecular sciences
Published: February 13, 2026
SKI regulates rRNA transcription and pericentromeric heterochromatin to ensure centromere integrity and genome stability.
Journal: Neoplasia (New York, N.Y.)
Published: March 26, 2025
ADAR-Mediated A>I(G) RNA Editing in the Genotoxic Drug Response of Breast Cancer.
Journal: International journal of molecular sciences
Published: May 04, 2024
A Comprehensive Analysis of the Effect of A>I(G) RNA-Editing Sites on Genotoxic Drug Response and Progression in Breast Cancer.
Journal: Biomedicines
Published: February 12, 2024
ADAR3 modulates neuronal differentiation and regulates mRNA stability and translation.
Journal: Nucleic acids research
Published: January 24, 2023
Detection of transcriptome-wide microRNA-target interactions in single cells with agoTRIBE.
Journal: Nature biotechnology
Published: August 26, 2022
ADAR1- and ADAR2-mediated regulation of maturation and targeting of miR-376b to modulate GABA neurotransmitter catabolism.
Journal: The Journal of biological chemistry
Published: October 19, 2021
Increase in ADAR1p110 activates the canonical Wnt signaling pathway associated with aggressive phenotype in triple negative breast cancer cells.
Journal: Gene
Published: July 20, 2021
ADAR1 Transcriptome editing promotes breast cancer progression through the regulation of cell cycle and DNA damage response.
Journal: Biochimica et biophysica acta. Molecular cell research
Published: October 27, 2019
Ski Is Required for Tri-Methylation of H3K9 in Major Satellite and for Repression of Pericentromeric Genes: Mmp3, Mmp10 and Mmp13, in Mouse Fibroblasts.
Journal: Journal of molecular biology
Published: October 24, 2019
Genotoxic stress triggers the activation of IRE1α-dependent RNA decay to modulate the DNA damage response.
Journal: Nature communications
Published: April 18, 2019
Complex I and II are required for normal mitochondrial Ca2+ homeostasis.
Journal: Mitochondrion
Published: August 18, 2018
Last Updated: 09/26/2026