Elena Britti

IRBLleida, Av. Rovira Roure, 80
Lleida, CT 25198, Spain

Last Updated: 09/26/2026

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Found 11 publications

Correction: Mitochondrial impairment, decreased Sirtuin activity and protein acetylation in dorsal root ganglia in Friedreich ataxia models.

Correction: Mitochondrial impairment, decreased Sirtuin activity and protein acetylation in dorsal root ganglia in Friedreich ataxia models.

Journal: Cellular and molecular life sciences : CMLS

Published: August 21, 2025

Mitochondrial impairment, decreased sirtuin activity and protein acetylation in dorsal root ganglia in Friedreich Ataxia models.

Mitochondrial impairment, decreased sirtuin activity and protein acetylation in dorsal root ganglia in Friedreich Ataxia models.

Journal: Cellular and molecular life sciences : CMLS

Published: May 17, 2023

Mice harboring the FXN I151F pathological point mutation present decreased frataxin levels, a Friedreich ataxia-like phenotype, and mitochondrial alterations.

Mice harboring the FXN I151F pathological point mutation present decreased frataxin levels, a Friedreich ataxia-like phenotype, and mitochondrial alterations.

Journal: Cellular and molecular life sciences : CMLS

Published: May 25, 2021

Mitochondrial iron and calcium homeostasis in Friedreich ataxia.

Mitochondrial iron and calcium homeostasis in Friedreich ataxia.

Journal: IUBMB life

Published: January 04, 2021

PPAR gamma agonist leriglitazone improves frataxin-loss impairments in cellular and animal models of Friedreich Ataxia.

PPAR gamma agonist leriglitazone improves frataxin-loss impairments in cellular and animal models of Friedreich Ataxia.

Journal: Neurobiology of disease

Published: August 06, 2020

Calcitriol increases frataxin levels and restores mitochondrial function in cell models of Friedreich Ataxia.

Calcitriol increases frataxin levels and restores mitochondrial function in cell models of Friedreich Ataxia.

Journal: The Biochemical journal

Published: May 04, 2020

Calpain-Inhibitors Protect Frataxin-Deficient Dorsal Root Ganglia Neurons from Loss of Mitochondrial Na+/Ca2+ Exchanger, NCLX, and Apoptosis.

Calpain-Inhibitors Protect Frataxin-Deficient Dorsal Root Ganglia Neurons from Loss of Mitochondrial Na+/Ca2+ Exchanger, NCLX, and Apoptosis.

Journal: Neurochemical research

Published: December 05, 2019

Tau inhibits mitochondrial calcium efflux and makes neurons vulnerable to calcium-induced cell death.

Tau inhibits mitochondrial calcium efflux and makes neurons vulnerable to calcium-induced cell death.

Journal: Cell calcium

Published: November 11, 2019

Mitochondrial calcium signalling and neurodegenerative diseases.

Mitochondrial calcium signalling and neurodegenerative diseases.

Journal: Neuronal signaling

Published: June 15, 2018

Mitochondrial pore opening and loss of Ca2+ exchanger NCLX levels occur after frataxin depletion.

Mitochondrial pore opening and loss of Ca2+ exchanger NCLX levels occur after frataxin depletion.

Journal: Biochimica et biophysica acta. Molecular basis of disease

Published: August 11, 2017

Frataxin-deficient neurons and mice models of Friedreich ataxia are improved by TAT-MTScs-FXN treatment.

Frataxin-deficient neurons and mice models of Friedreich ataxia are improved by TAT-MTScs-FXN treatment.

Journal: Journal of cellular and molecular medicine

Published: April 10, 2017

Showing 1-11 of 11

Last Updated: 09/26/2026