Eliana Trapani
Turin, Italy
Last Updated: 09/26/2026
Save publications for later
Sign Up
Not sure about your diagnosis?
Check Your Symptoms
Found 10 publications
KRIT1 loss-of-function induces a chronic Nrf2-mediated adaptive homeostasis that sensitizes cells to oxidative stress: Implication for Cerebral Cavernous Malformation disease.
Journal: Free radical biology & medicine
Published: March 26, 2017
Up-regulation of NADPH oxidase-mediated redox signaling contributes to the loss of barrier function in KRIT1 deficient endothelium.
Journal: Scientific reports
Published: March 13, 2017
Beyond multiple mechanisms and a unique drug: Defective autophagy as pivotal player in cerebral cavernous malformation pathogenesis and implications for targeted therapies.
Journal: Rare diseases (Austin, Tex.)
Published: November 17, 2015
Cytochrome P450 and matrix metalloproteinase genetic modifiers of disease severity in Cerebral Cavernous Malformation type 1.
Journal: Free radical biology & medicine
Published: November 12, 2015
Defective autophagy is a key feature of cerebral cavernous malformations.
Journal: EMBO molecular medicine
Published: September 30, 2015
Cerebral cavernous malformation (CCM) disease: from monogenic forms to genetic susceptibility factors.
Journal: Journal of neurosurgical sciences
Published: April 22, 2015
Evaluation of the bioactive properties of avenanthramide analogs produced in recombinant yeast.
Journal: BioFactors (Oxford, England)
Published: October 09, 2014
The Ras superfamily of small GTPases: the unlocked secrets.
Journal: Methods in molecular biology (Clifton, N.J.)
Published: January 29, 2014
KRIT1 loss of function causes a ROS-dependent upregulation of c-Jun.
Journal: Free radical biology & medicine
Published: July 11, 2013
Showing 1-10 of 10
Last Updated: 09/26/2026