Elina Kats
Carl Neuberg Str. 1
Hannover, NI 30625, Germany
Hannover, NI 30625, Germany
Last Updated: 09/26/2026
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Found 8 publications
The monosialoganglioside GM1a protects against complement attack.
Journal: Cell death discovery
Published: June 06, 2023
Uridine diphosphate-N-acetylglucosamine-2-epimerase/N-acetylmannosamine kinase deletion in mice leads to lethal intracerebral hemorrhage during embryonic development.
Journal: Glycobiology
Published: May 20, 2021
Podocyte-Specific Sialylation-Deficient Mice Serve as a Model for Human FSGS.
Journal: Journal of the American Society of Nephrology : JASN
Published: September 25, 2018
Sialic acid is a critical fetal defense against maternal complement attack.
Journal: The Journal of clinical investigation
Published: January 19, 2018
Sialylation Is Dispensable for Early Murine Embryonic Development in Vitro.
Journal: Chembiochem : a European journal of chemical biology
Published: February 17, 2017
Deficits in sialylation impair podocyte maturation.
Journal: Journal of the American Society of Nephrology : JASN
Published: June 30, 2012
Identification and biochemical characterization of two functional CMP-sialic acid synthetases in Danio rerio.
Journal: The Journal of biological chemistry
Published: February 22, 2012
Showing 1-8 of 8
Last Updated: 09/26/2026