Elisa Cabiscol

Av Rovira Roure 80
Lleida, CT 25198, Spain

Last Updated: 09/26/2026

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Found 25 publications

Targeting frataxin deficiency in DRG neurons and fibroblasts: omaveloxolone restores metabolic and iron balance to reduce ferroptosis.

Targeting frataxin deficiency in DRG neurons and fibroblasts: omaveloxolone restores metabolic and iron balance to reduce ferroptosis.

Journal: Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie

Published: December 03, 2025

Endogenous mitochondrial hydrogen peroxide regulates neurogenesis during cortical development.

Endogenous mitochondrial hydrogen peroxide regulates neurogenesis during cortical development.

Journal: Redox biology

Published: October 06, 2025

Correction: Mitochondrial impairment, decreased Sirtuin activity and protein acetylation in dorsal root ganglia in Friedreich ataxia models.

Correction: Mitochondrial impairment, decreased Sirtuin activity and protein acetylation in dorsal root ganglia in Friedreich ataxia models.

Journal: Cellular and molecular life sciences : CMLS

Published: August 21, 2025

Leriglitazone improves iron homeostasis and ferroptotic markers in frataxin-deficient dorsal root ganglia neurons.

Leriglitazone improves iron homeostasis and ferroptotic markers in frataxin-deficient dorsal root ganglia neurons.

Journal: Biomedicine & pharmacotherapy = Biomedecine & pharmacotherapie

Published: July 08, 2025

Pathological frataxin deficiency in mice causes tissue-specific alterations in iron homeostasis.

Pathological frataxin deficiency in mice causes tissue-specific alterations in iron homeostasis.

Journal: iScience

Published: March 28, 2025

Corrigendum to "Deciphering the ferroptosis pathways in dorsal root ganglia of Friedreich ataxia models. The role of LKB1/AMPK, KEAP1, and GSK3β in the impairment of the NRF2 response" [Redox Biol. 76 (2024) 103339].

Corrigendum to "Deciphering the ferroptosis pathways in dorsal root ganglia of Friedreich ataxia models. The role of LKB1/AMPK, KEAP1, and GSK3β in the impairment of the NRF2 response" [Redox Biol. 76 (2024) 103339].

Journal: Redox biology

Published: March 08, 2025

Deciphering the ferroptosis pathways in dorsal root ganglia of Friedreich ataxia models. The role of LKB1/AMPK, KEAP1, and GSK3β in the impairment of the NRF2 response.

Deciphering the ferroptosis pathways in dorsal root ganglia of Friedreich ataxia models. The role of LKB1/AMPK, KEAP1, and GSK3β in the impairment of the NRF2 response.

Journal: Redox biology

Published: August 02, 2024

Reply to: "Calcitriol in Friedreich Ataxia".

Reply to: "Calcitriol in Friedreich Ataxia".

Journal: Movement disorders : official journal of the Movement Disorder Society

Published: July 22, 2024

Calcitriol Treatment Is Safe and Increases Frataxin Levels in Friedreich Ataxia Patients.

Calcitriol Treatment Is Safe and Increases Frataxin Levels in Friedreich Ataxia Patients.

Journal: Movement disorders : official journal of the Movement Disorder Society

Published: December 06, 2023

Mitochondrial impairment, decreased sirtuin activity and protein acetylation in dorsal root ganglia in Friedreich Ataxia models.

Mitochondrial impairment, decreased sirtuin activity and protein acetylation in dorsal root ganglia in Friedreich Ataxia models.

Journal: Cellular and molecular life sciences : CMLS

Published: May 17, 2023

Oxidative stress-induced FAK activation contributes to uterine serous carcinoma aggressiveness.

Oxidative stress-induced FAK activation contributes to uterine serous carcinoma aggressiveness.

Journal: Molecular oncology

Published: September 12, 2022

Mice harboring the FXN I151F pathological point mutation present decreased frataxin levels, a Friedreich ataxia-like phenotype, and mitochondrial alterations.

Mice harboring the FXN I151F pathological point mutation present decreased frataxin levels, a Friedreich ataxia-like phenotype, and mitochondrial alterations.

Journal: Cellular and molecular life sciences : CMLS

Published: May 25, 2021

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Last Updated: 09/26/2026