Emi Nose
Hongo, 1138655, Japan
Last Updated: 09/26/2026
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Found 8 publications
Notch Signaling Induced by Endoplasmic Reticulum Stress Regulates Cumulus-Oocyte Complex Expansion in Polycystic Ovary Syndrome.
Journal: Biomolecules
Published: May 30, 2022
Induction of aryl hydrocarbon receptor in granulosa cells by endoplasmic reticulum stress contributes to pathology of polycystic ovary syndrome.
Journal: Molecular human reproduction
Published: November 08, 2020
Activation of endoplasmic reticulum stress mediates oxidative stress-induced apoptosis of granulosa cells in ovaries affected by endometrioma.
Journal: Molecular human reproduction
Published: August 29, 2019
Accumulation of advanced glycation end products in follicles is associated with poor oocyte developmental competence.
Journal: Molecular human reproduction
Published: March 04, 2019
Endoplasmic Reticulum Stress Activated by Androgen Enhances Apoptosis of Granulosa Cells via Induction of Death Receptor 5 in PCOS.
Journal: Endocrinology
Published: July 20, 2018
Activation of Endoplasmic Reticulum Stress in Granulosa Cells from Patients with Polycystic Ovary Syndrome Contributes to Ovarian Fibrosis.
Journal: Scientific reports
Published: May 16, 2017
Evidence of the activation of unfolded protein response in granulosa and cumulus cells during follicular growth and maturation.
Journal: Gynecological endocrinology : the official journal of the International Society of Gynecological Endocrinology
Published: August 21, 2015
Showing 1-8 of 8
Last Updated: 09/26/2026