Emily B. Pratt

Pediatrics

445 East Main Street
Hillsboro, OR 97123

Last Updated: 09/26/2026

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Found 2 publications

Engineered Kir6.2 mutations that correct the trafficking defect of K(ATP) channels caused by specific SUR1 mutations.

Engineered Kir6.2 mutations that correct the trafficking defect of K(ATP) channels caused by specific SUR1 mutations.

Journal: Channels (Austin, Tex.)

Published: May 23, 2013

Engineered interaction between SUR1 and Kir6.2 that enhances ATP sensitivity in KATP channels.

Engineered interaction between SUR1 and Kir6.2 that enhances ATP sensitivity in KATP channels.

Journal: The Journal of general physiology

Published: July 18, 2012

Showing 1-2 of 2

Last Updated: 09/26/2026