Enno Klussmann

Berlin, BE, Germany

Last Updated: 09/26/2026

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Found 64 publications

AKAP signaling: physiological and pathophysiological roles and opportunities for novel therapeutic concepts.

AKAP signaling: physiological and pathophysiological roles and opportunities for novel therapeutic concepts.

Journal: Physiological reviews

Published: May 19, 2026

Can MQ232 guide to novel options for treatment of ADPKD?

Can MQ232 guide to novel options for treatment of ADPKD?

Journal: Kidney international

Published: May 12, 2026

Synthesis and characterization of cortinarins - cryptic cyclic peptides from mushrooms of the genus Cortinarius.

Synthesis and characterization of cortinarins - cryptic cyclic peptides from mushrooms of the genus Cortinarius.

Journal: Chemical science

Published: March 17, 2026

Live-cell quantitative monitoring reveals distinct, high-affinity Gβγ regulations of GIRK2 and GIRK1/2 channels.

Live-cell quantitative monitoring reveals distinct, high-affinity Gβγ regulations of GIRK2 and GIRK1/2 channels.

Journal: bioRxiv : the preprint server for biology

Published: November 24, 2025

Live-cell quantitative monitoring reveals distinct, high-affinity Gβγ regulations of GIRK2 and GIRK1/2 channels.

Live-cell quantitative monitoring reveals distinct, high-affinity Gβγ regulations of GIRK2 and GIRK1/2 channels.

Journal: Nature communications

Published: March 30, 2025

PDE3A as a Therapeutic Target for the Modulation of Compartmentalised Cyclic Nucleotide-Dependent Signalling.

PDE3A as a Therapeutic Target for the Modulation of Compartmentalised Cyclic Nucleotide-Dependent Signalling.

Journal: Cells

Published: March 28, 2025

Withdrawal: RNA Silencing Identifies PDE4D5 as the Functionally Relevant cAMP Phosphodiesterase Interacting with βArrestin to Control the Protein Kinase A/AKAP79-mediated Switching of the β2-Adrenergic Receptor to Activation of ERK in HEK293B2 Cells.

Withdrawal: RNA Silencing Identifies PDE4D5 as the Functionally Relevant cAMP Phosphodiesterase Interacting with βArrestin to Control the Protein Kinase A/AKAP79-mediated Switching of the β2-Adrenergic Receptor to Activation of ERK in HEK293B2 Cells.

Journal: The Journal of biological chemistry

Published: February 17, 2025

Activated CaMKIIδ translocates to the RyR nanodomain in cardiomyocytes.

Activated CaMKIIδ translocates to the RyR nanodomain in cardiomyocytes.

Journal: Cardiovascular research

Published: October 04, 2024

Tripartite interactions of PKA catalytic subunit and C-terminal domains of cardiac Ca2+ channel may modulate its β-adrenergic regulation.

Tripartite interactions of PKA catalytic subunit and C-terminal domains of cardiac Ca2+ channel may modulate its β-adrenergic regulation.

Journal: BMC biology

Published: November 01, 2023

Mutations in Phosphodiesterase 3A (PDE3A) Cause Hypertension Without Cardiac Damage.

Mutations in Phosphodiesterase 3A (PDE3A) Cause Hypertension Without Cardiac Damage.

Journal: Hypertension (Dallas, Tex. : 1979)

Published: April 10, 2023

Many kinases for controlling the water channel aquaporin-2.

Many kinases for controlling the water channel aquaporin-2.

Journal: The Journal of physiology

Published: March 29, 2023

Integrated Proteomics Unveils Nuclear PDE3A2 as a Regulator of Cardiac Myocyte Hypertrophy.

Integrated Proteomics Unveils Nuclear PDE3A2 as a Regulator of Cardiac Myocyte Hypertrophy.

Journal: Circulation research

Published: March 08, 2023

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Last Updated: 09/26/2026