Erdenebelig Uyangaa
Iksan, Republic of Korea
Last Updated: 09/26/2026
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Found 15 publications
Myeloid miR-155 deficiency exacerbates viral encephalitis by hindering M1 macrophage polarization due to impaired NLRP3 inflammasome activation in extraneural tissues.
Journal: Frontiers in immunology
Published: February 26, 2026
T cell-intrinsic miR-155 is required for Th2 and Th17-biased responses in acute and chronic airway inflammation by targeting several different transcription factors.
Journal: Immunology
Published: October 30, 2021
Indoleamine 2,3-Dioxygenase in Hematopoietic Stem Cell-Derived Cells Suppresses Rhinovirus-Induced Neutrophilic Airway Inflammation by Regulating Th1- and Th17-Type Responses.
Journal: Immune network
Published: June 25, 2021
Type I IFN signaling limits hemorrhage-like disease after infection with Japanese encephalitis virus through modulating a prerequisite infection of CD11b+Ly-6C+ monocytes.
Journal: Journal of neuroinflammation
Published: December 04, 2020
Double-faced implication of CD4+ Foxp3+ regulatory T cells expanded by acute dengue infection via TLR2/MyD88 pathway.
Journal: European journal of immunology
Published: September 24, 2019
CCR5 attenuates neutrophilic airway inflammation exacerbated by infection with rhinovirus.
Journal: Cellular immunology
Published: August 17, 2019
Indispensable Role of CX3CR1+ Dendritic Cells in Regulation of Virus-Induced Neuroinflammation Through Rapid Development of Antiviral Immunity in Peripheral Lymphoid Tissues.
Journal: Frontiers in immunology
Published: November 15, 2018
TLR2/MyD88 pathway-dependent regulation of dendritic cells by dengue virus promotes antibody-dependent enhancement via Th2-biased immunity.
Journal: Oncotarget
Published: July 11, 2017
Exacerbation of Japanese Encephalitis by CD11chi Dendritic Cell Ablation Is Associated with an Imbalance in Regulatory Foxp3+ and IL-17+CD4+ Th17 Cells and in Ly-6Chi and Ly-6Clo Monocytes.
Journal: Immune network
Published: February 27, 2017
Ablation of CD11c(hi) dendritic cells exacerbates Japanese encephalitis by regulating blood-brain barrier permeability and altering tight junction/adhesion molecules.
Journal: Comparative immunology, microbiology and infectious diseases
Published: April 01, 2016
CCR5 ameliorates Japanese encephalitis via dictating the equilibrium of regulatory CD4(+)Foxp3(+) T and IL-17(+)CD4(+) Th17 cells.
Journal: Journal of neuroinflammation
Published: December 28, 2015
Amelioration of Japanese encephalitis by blockage of 4-1BB signaling is coupled to divergent enhancement of type I/II IFN responses and Ly-6C(hi) monocyte differentiation.
Journal: Journal of neuroinflammation
Published: October 02, 2015
Last Updated: 09/26/2026