Eugenia Kress

Aachen, NW, Germany

Last Updated: 09/26/2026

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Found 12 publications

Lack of chemokine (C-C motif) ligand 3 leads to decreased survival and reduced immune response after bacterial meningitis.

Lack of chemokine (C-C motif) ligand 3 leads to decreased survival and reduced immune response after bacterial meningitis.

Journal: Cytokine

Published: March 23, 2018

Toll-Like Receptor 2-Mediated Glial Cell Activation in a Mouse Model of Cuprizone-Induced Demyelination.

Toll-Like Receptor 2-Mediated Glial Cell Activation in a Mouse Model of Cuprizone-Induced Demyelination.

Journal: Molecular neurobiology

Published: July 18, 2017

CRAMP deficiency leads to a pro-inflammatory phenotype and impaired phagocytosis after exposure to bacterial meningitis pathogens.

CRAMP deficiency leads to a pro-inflammatory phenotype and impaired phagocytosis after exposure to bacterial meningitis pathogens.

Journal: Cell communication and signaling : CCS

Published: May 08, 2017

Combination of cuprizone and experimental autoimmune encephalomyelitis to study inflammatory brain lesion formation and progression.

Combination of cuprizone and experimental autoimmune encephalomyelitis to study inflammatory brain lesion formation and progression.

Journal: Glia

Published: April 03, 2017

Formyl Peptide Receptor 1-Mediated Glial Cell Activation in a Mouse Model of Cuprizone-Induced Demyelination.

Formyl Peptide Receptor 1-Mediated Glial Cell Activation in a Mouse Model of Cuprizone-Induced Demyelination.

Journal: Journal of molecular neuroscience : MN

Published: March 10, 2017

Lack of Toll-like receptor 2 results in higher mortality of bacterial meningitis by impaired host resistance.

Lack of Toll-like receptor 2 results in higher mortality of bacterial meningitis by impaired host resistance.

Journal: Journal of neuroimmunology

Published: May 12, 2016

Psoriasin has divergent effects on the innate immune responses of murine glial cells.

Psoriasin has divergent effects on the innate immune responses of murine glial cells.

Journal: Journal of neurochemistry

Published: April 15, 2016

Lack of Proinflammatory Cytokine Interleukin-6 or Tumor Necrosis Factor Receptor-1 Results in a Failure of the Innate Immune Response after Bacterial Meningitis.

Lack of Proinflammatory Cytokine Interleukin-6 or Tumor Necrosis Factor Receptor-1 Results in a Failure of the Innate Immune Response after Bacterial Meningitis.

Journal: Mediators of inflammation

Published: November 13, 2015

Intrathecal application of the antimicrobial peptide CRAMP reduced mortality and neuroinflammation in an experimental model of pneumococcal meningitis.

Intrathecal application of the antimicrobial peptide CRAMP reduced mortality and neuroinflammation in an experimental model of pneumococcal meningitis.

Journal: The Journal of infection

Published: February 04, 2015

Lack of formyl peptide receptor 1 and 2 leads to more severe inflammation and higher mortality in mice with of pneumococcal meningitis.

Lack of formyl peptide receptor 1 and 2 leads to more severe inflammation and higher mortality in mice with of pneumococcal meningitis.

Journal: Immunology

Published: January 31, 2014

Role of the cathelicidin-related antimicrobial peptide in inflammation and mortality in a mouse model of bacterial meningitis.

Role of the cathelicidin-related antimicrobial peptide in inflammation and mortality in a mouse model of bacterial meningitis.

Journal: Journal of innate immunity

Published: April 08, 2013

The anatomy of non-carious cervical lesions.

The anatomy of non-carious cervical lesions.

Journal: Clinical oral investigations

Published: September 24, 2012

Showing 1-12 of 12

Last Updated: 09/26/2026