Eun H. Lee
Seoul, Republic of Korea
Last Updated: 09/26/2026
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Found 30 publications
Defects in skeletal myotubes caused by STIM1 I115F that lead to tubular aggregate myopathy and Stormorken syndrome, and their restoration at the cellular level.
Journal: American journal of physiology. Cell physiology
Published: April 15, 2026
Tripartite Motif-Containing Protein 32 (TRIM32): What Does It Do for Skeletal Muscle?
Journal: Cells
Published: July 19, 2023
Tripartite motif-containing protein 32 regulates Ca2+ movement in skeletal muscle.
Journal: American journal of physiology. Cell physiology
Published: November 14, 2022
Calsequestrin 1 Is an Active Partner of Stromal Interaction Molecule 2 in Skeletal Muscle.
Journal: Cells
Published: September 08, 2021
Calsequestrin: a well-known but curious protein in skeletal muscle.
Journal: Experimental & molecular medicine
Published: September 02, 2020
A muscular hypotonia-associated STIM1 mutant at R429 induces abnormalities in intracellular Ca2+ movement and extracellular Ca2+ entry in skeletal muscle.
Journal: Scientific reports
Published: September 11, 2019
Propranolol increases vascular permeability through pericyte apoptosis and exacerbates oxygen-induced retinopathy.
Journal: Biochemical and biophysical research communications
Published: July 17, 2018
Oleanolic Acids Inhibit Vascular Endothelial Growth Factor Receptor 2 Signaling in Endothelial Cells: Implication for Anti-Angiogenic Therapy.
Journal: Molecules and cells
Published: May 14, 2018
Retinal pigment epithelium-derived transforming growth factor-β2 inhibits the angiogenic response of endothelial cells by decreasing vascular endothelial growth factor receptor-2 expression.
Journal: Journal of cellular physiology
Published: April 30, 2018
Last Updated: 09/26/2026