Eva M. Szego

Bonn, NW 53127, Germany

Last Updated: 09/26/2026

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Found 34 publications

Metabolic reprogramming and altered ATP content impair neuroprotective functions of microglia in β-glucocerebrosidase deficiency models.

Metabolic reprogramming and altered ATP content impair neuroprotective functions of microglia in β-glucocerebrosidase deficiency models.

Journal: Journal of neuroinflammation

Published: May 30, 2025

Intermittent fasting reduces alpha-synuclein pathology and functional decline in a mouse model of Parkinson's disease.

Intermittent fasting reduces alpha-synuclein pathology and functional decline in a mouse model of Parkinson's disease.

Journal: Nature communications

Published: February 24, 2025

Tissue inflammation induced by constitutively active STING is mediated by enhanced TNF signaling.

Tissue inflammation induced by constitutively active STING is mediated by enhanced TNF signaling.

Journal: eLife

Published: July 08, 2024

Mitochondrial oxidant stress promotes α-synuclein aggregation and spreading in mice with mutated glucocerebrosidase.

Mitochondrial oxidant stress promotes α-synuclein aggregation and spreading in mice with mutated glucocerebrosidase.

Journal: NPJ Parkinson's disease

Published: June 28, 2024

Evaluation of the Effect of β-Wrapin AS69 in a Mouse Model Based on Alpha-Synuclein Overexpression.

Evaluation of the Effect of β-Wrapin AS69 in a Mouse Model Based on Alpha-Synuclein Overexpression.

Journal: Biomolecules

Published: May 01, 2024

Axonal Lysosomal Assays for Characterizing the Effects of LRRK2 G2019S.

Axonal Lysosomal Assays for Characterizing the Effects of LRRK2 G2019S.

Journal: Biology

Published: November 10, 2023

Constitutively active STING causes neuroinflammation and degeneration of dopaminergic neurons in mice.

Constitutively active STING causes neuroinflammation and degeneration of dopaminergic neurons in mice.

Journal: eLife

Published: August 02, 2022

Rab7 reduces α-synuclein toxicity in rats and primary neurons.

Rab7 reduces α-synuclein toxicity in rats and primary neurons.

Journal: Experimental neurology

Published: July 12, 2021

Erratum to: Glycation potentiates α-synuclein-associated neurodegeneration in synucleinopathies.

Erratum to: Glycation potentiates α-synuclein-associated neurodegeneration in synucleinopathies.

Journal: Brain : a journal of neurology

Published: June 08, 2021

A β-Wrapin Targeting the N-Terminus of α-Synuclein Monomers Reduces Fibril-Induced Aggregation in Neurons.

A β-Wrapin Targeting the N-Terminus of α-Synuclein Monomers Reduces Fibril-Induced Aggregation in Neurons.

Journal: Frontiers in neuroscience

Published: April 16, 2021

Maturing Autophagosomes are Transported Towards the Cell Periphery.

Maturing Autophagosomes are Transported Towards the Cell Periphery.

Journal: Cellular and molecular neurobiology

Published: December 30, 2020

Cytosolic Trapping of a Mitochondrial Heat Shock Protein Is an Early Pathological Event in Synucleinopathies.

Cytosolic Trapping of a Mitochondrial Heat Shock Protein Is an Early Pathological Event in Synucleinopathies.

Journal: Cell reports

Published: November 23, 2018

Showing 1-12 of 34

Last Updated: 09/26/2026