Evgeny E. Akkuratov

Solna, AB 17121, Sweden

Last Updated: 09/26/2026

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Found 18 publications

ATP1A3 dysfunction causes motor hyperexcitability and afterhyperpolarization loss in a dystonia model.

ATP1A3 dysfunction causes motor hyperexcitability and afterhyperpolarization loss in a dystonia model.

Journal: Brain : a journal of neurology
Published: May 17, 2024

Detection and quantification of Na,K-ATPase dimers in the plasma membrane of living cells by FRET-FCS.

Detection and quantification of Na,K-ATPase dimers in the plasma membrane of living cells by FRET-FCS.

Journal: Biochimica et biophysica acta. General subjects
Published: January 04, 2024

Author Correction: A previously uncharacterized Factor Associated with Metabolism and Energy (FAME/C14orf105/CCDC198/1700011H14Rik) is related to evolutionary adaptation, energy balance, and kidney physiology.

Author Correction: A previously uncharacterized Factor Associated with Metabolism and Energy (FAME/C14orf105/CCDC198/1700011H14Rik) is related to evolutionary adaptation, energy balance, and kidney physiology.

Journal: Nature communications
Published: June 15, 2023

MindReader: Unsupervised Classification of Electroencephalographic Data.

MindReader: Unsupervised Classification of Electroencephalographic Data.

Journal: Sensors (Basel, Switzerland)
Published: January 25, 2023

A previously uncharacterized Factor Associated with Metabolism and Energy (FAME/C14orf105/CCDC198/1700011H14Rik) is related to evolutionary adaptation, energy balance, and kidney physiology.

A previously uncharacterized Factor Associated with Metabolism and Energy (FAME/C14orf105/CCDC198/1700011H14Rik) is related to evolutionary adaptation, energy balance, and kidney physiology.

Journal: Nature communications
Published: September 24, 2021

A missense mutation converts the Na+,K+-ATPase into an ion channel and causes therapy-resistant epilepsy.

A missense mutation converts the Na+,K+-ATPase into an ion channel and causes therapy-resistant epilepsy.

Journal: The Journal of biological chemistry
Published: May 04, 2021

Ouabain-Induced Gene Expression Changes in Human iPSC-Derived Neuron Culture Expressing Dopamine and cAMP-Regulated Phosphoprotein 32 and GABA Receptors.

Ouabain-Induced Gene Expression Changes in Human iPSC-Derived Neuron Culture Expressing Dopamine and cAMP-Regulated Phosphoprotein 32 and GABA Receptors.

Journal: Brain sciences
Published: December 21, 2020

Ouabain Modulates the Functional Interaction Between Na,K-ATPase and NMDA Receptor.

Ouabain Modulates the Functional Interaction Between Na,K-ATPase and NMDA Receptor.

Journal: Molecular neurobiology
Published: January 27, 2020

Chlamydia pan-genomic analysis reveals balance between host adaptation and selective pressure to genome reduction.

Chlamydia pan-genomic analysis reveals balance between host adaptation and selective pressure to genome reduction.

Journal: BMC genomics
Published: February 14, 2019

Neanderthal and Denisovan ancestry in Papuans: A functional study.

Neanderthal and Denisovan ancestry in Papuans: A functional study.

Journal: Journal of bioinformatics and computational biology
Published: May 10, 2018

Comparative Action of Cardiotonic Steroids on Intracellular Processes in Rat Cortical Neurons.

Comparative Action of Cardiotonic Steroids on Intracellular Processes in Rat Cortical Neurons.

Journal: Biochemistry. Biokhimiia
Published: April 06, 2018

Error-prone bypass of DNA lesions during lagging-strand replication is a common source of germline and cancer mutations.

Error-prone bypass of DNA lesions during lagging-strand replication is a common source of germline and cancer mutations.

Journal: Nature genetics
Published: October 27, 2017
Showing 1-12 of 18

Last Updated: 09/26/2026