Ezra B. Ketema

Edmonton, AB, Canada

Last Updated: 09/26/2026

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Found 13 publications

Loss of SIRT2 in cardiomyocytes reduces glycolytic flux via increased acetylation of glycolytic enzymes.

Loss of SIRT2 in cardiomyocytes reduces glycolytic flux via increased acetylation of glycolytic enzymes.

Journal: American journal of physiology. Heart and circulatory physiology
Published: June 01, 2026

Lowering Cardiac Branched-Chain Keto Acid Levels Enhances Cardiac Glucose Oxidation and Cardiac Efficiency via Enhancing Mitochondrial Insulin Signaling in Heart Failure.

Lowering Cardiac Branched-Chain Keto Acid Levels Enhances Cardiac Glucose Oxidation and Cardiac Efficiency via Enhancing Mitochondrial Insulin Signaling in Heart Failure.

Journal: Circulation. Heart failure
Published: July 25, 2025

Alterations of myocardial ketone metabolism in heart failure with preserved ejection fraction (HFpEF).

Alterations of myocardial ketone metabolism in heart failure with preserved ejection fraction (HFpEF).

Journal: ESC heart failure
Published: January 28, 2025

The Impact of Obesity on Cardiac Energy Metabolism and Efficiency in Heart Failure With Preserved Ejection Fraction.

The Impact of Obesity on Cardiac Energy Metabolism and Efficiency in Heart Failure With Preserved Ejection Fraction.

Journal: The Canadian journal of cardiology
Published: November 27, 2024

Glycolysis in heart failure with preserved ejection fraction.

Glycolysis in heart failure with preserved ejection fraction.

Journal: European journal of heart failure
Published: August 02, 2024

The role of acetylation in obesity-induced cardiac metabolic alterations.

The role of acetylation in obesity-induced cardiac metabolic alterations.

Journal: Journal of pharmacy & pharmaceutical sciences : a publication of the Canadian Society for Pharmaceutical Sciences, Societe canadienne des sciences pharmaceutiques
Published: April 02, 2024

Protein lysine acetylation does not contribute to the high rates of fatty acid oxidation seen in the post-ischemic heart.

Protein lysine acetylation does not contribute to the high rates of fatty acid oxidation seen in the post-ischemic heart.

Journal: Scientific reports
Published: October 20, 2023

Mitochondrial fatty acid oxidation is the major source of cardiac adenosine triphosphate production in heart failure with preserved ejection fraction.

Mitochondrial fatty acid oxidation is the major source of cardiac adenosine triphosphate production in heart failure with preserved ejection fraction.

Journal: Cardiovascular research
Published: August 31, 2023

Corrigendum to: Concurrent diabetes and heart failure: interplay and novel therapeutic approaches.

Corrigendum to: Concurrent diabetes and heart failure: interplay and novel therapeutic approaches.

Journal: Cardiovascular research
Published: December 13, 2021

Metabolic, structural and biochemical changes in diabetes and the development of heart failure.

Metabolic, structural and biochemical changes in diabetes and the development of heart failure.

Journal: Diabetologia
Published: July 28, 2021

Post-translational Acetylation Control of Cardiac Energy Metabolism.

Post-translational Acetylation Control of Cardiac Energy Metabolism.

Journal: Frontiers in cardiovascular medicine
Published: June 11, 2021

Concurrent diabetes and heart failure: interplay and novel therapeutic approaches.

Concurrent diabetes and heart failure: interplay and novel therapeutic approaches.

Journal: Cardiovascular research
Published: December 18, 2020
Showing 1-12 of 13

Last Updated: 09/26/2026