Fabiane Sonego

Ribeirao Preto, SP, Brazil

Last Updated: 09/26/2026

Save publications for later

Sign Up

Not sure about your diagnosis?

Check Your Symptoms

Found 13 publications

Gasdermin D inhibition prevents multiple organ dysfunction during sepsis by blocking NET formation.

Gasdermin D inhibition prevents multiple organ dysfunction during sepsis by blocking NET formation.

Journal: Blood

Published: March 02, 2021

CCR5-Positive Inflammatory Monocytes are Crucial for Control of Sepsis.

CCR5-Positive Inflammatory Monocytes are Crucial for Control of Sepsis.

Journal: Shock (Augusta, Ga.)

Published: February 07, 2019

The Atypical Chemokine Receptor ACKR2 is Protective Against Sepsis.

The Atypical Chemokine Receptor ACKR2 is Protective Against Sepsis.

Journal: Shock (Augusta, Ga.)

Published: March 29, 2018

The aggravation of arthritis by periodontitis is dependent of IL-17 receptor A activation.

The aggravation of arthritis by periodontitis is dependent of IL-17 receptor A activation.

Journal: Journal of clinical periodontology

Published: May 08, 2017

The host control of a clinical isolate strain of P. aeruginosa infection is independent of Nod-1 but depends on MyD88.

The host control of a clinical isolate strain of P. aeruginosa infection is independent of Nod-1 but depends on MyD88.

Journal: Inflammation research : official journal of the European Histamine Research Society ... [et al.]

Published: December 19, 2016

Therapeutic potential and limitations of cholinergic anti-inflammatory pathway in sepsis.

Therapeutic potential and limitations of cholinergic anti-inflammatory pathway in sepsis.

Journal: Pharmacological research

Published: November 07, 2016

Paradoxical Roles of the Neutrophil in Sepsis: Protective and Deleterious.

Paradoxical Roles of the Neutrophil in Sepsis: Protective and Deleterious.

Journal: Frontiers in immunology

Published: February 19, 2016

Neutrophil Extracellular Traps Induce Organ Damage during Experimental and Clinical Sepsis.

Neutrophil Extracellular Traps Induce Organ Damage during Experimental and Clinical Sepsis.

Journal: PloS one

Published: September 08, 2015

Targeting neutrophils in sepsis.

Targeting neutrophils in sepsis.

Journal: Expert review of clinical immunology

Published: May 29, 2014

PPAR-γ/IL-10 axis inhibits MyD88 expression and ameliorates murine polymicrobial sepsis.

PPAR-γ/IL-10 axis inhibits MyD88 expression and ameliorates murine polymicrobial sepsis.

Journal: Journal of immunology (Baltimore, Md. : 1950)

Published: February 04, 2014

MyD88-, but not Nod1- and/or Nod2-deficient mice, show increased susceptibility to polymicrobial sepsis due to impaired local inflammatory response.

MyD88-, but not Nod1- and/or Nod2-deficient mice, show increased susceptibility to polymicrobial sepsis due to impaired local inflammatory response.

Journal: PloS one

Published: July 29, 2013

Heme oxygenase inhibition enhances neutrophil migration into the bronchoalveolar spaces and improves the outcome of murine pneumonia-induced sepsis.

Heme oxygenase inhibition enhances neutrophil migration into the bronchoalveolar spaces and improves the outcome of murine pneumonia-induced sepsis.

Journal: Shock (Augusta, Ga.)

Published: March 14, 2013

Showing 1-12 of 13

Last Updated: 09/26/2026