Fabricio M. Almeida
Campos Dos Goytacazes, RJ, Brazil
Last Updated: 09/26/2026
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Found 10 publications
CD39 regulates P2RX7-mediated lung necrotic lesions in severe experimental tuberculosis.
Journal: Mucosal immunology
Published: July 15, 2025
A Murine Model of Mycobacterium kansasii Infection Reproducing Necrotic Lung Pathology Reveals Considerable Heterogeneity in Virulence of Clinical Isolates.
Journal: Frontiers in microbiology
Published: May 31, 2021
Harmful Effects of Granulocytic Myeloid-Derived Suppressor Cells on Tuberculosis Caused by Hypervirulent Mycobacteria.
Journal: The Journal of infectious diseases
Published: May 26, 2020
Inhibiting Adenosine Receptor Signaling Promotes Accumulation of Effector CD4+ T Cells in the Lung Parenchyma During Severe Tuberculosis.
Journal: The Journal of infectious diseases
Published: February 12, 2018
Hypervirulent Mycobacterium tuberculosis strain triggers necrotic lung pathology associated with enhanced recruitment of neutrophils in resistant C57BL/6 mice.
Journal: PloS one
Published: August 26, 2016
Mycobacterium tuberculosis strains of the modern sublineage of the Beijing family are more likely to display increased virulence than strains of the ancient sublineage.
Journal: Journal of clinical microbiology
Published: May 16, 2014
Pulmonary infection with hypervirulent Mycobacteria reveals a crucial role for the P2X7 receptor in aggressive forms of tuberculosis.
Journal: PLoS pathogens
Published: March 08, 2013
Pathogenic Mycobacterium bovis strains differ in their ability to modulate the proinflammatory activation phenotype of macrophages.
Journal: BMC microbiology
Published: March 08, 2012
Showing 1-10 of 10
Last Updated: 09/26/2026