Fanny Eysert
660 Route Des Lucioles, Sophia Antipolis
Valbonne, 06560, France
Valbonne, 06560, France
Last Updated: 09/26/2026
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Found 15 publications
Peripheral alterations of mitochondrial function and mitophagy in Alzheimer's disease: from fundamental research to clinical perspectives.
Journal: Current opinion in neurology
Published: February 02, 2026
Inflammatory stimulus enhances synaptic material uptake by adult APP microglia in a microfluidic neuron-microglia co-culture model.
Journal: Journal of neuroinflammation
Published: September 05, 2025
Hampered AMPK-ULK1 cascade in Alzheimer's disease (AD) instigates mitochondria dysfunctions and AD-related alterations which are alleviated by metformin.
Journal: Alzheimer's research & therapy
Published: March 06, 2025
Mitochondrial alterations in fibroblasts from sporadic Alzheimer's disease (AD) patients correlate with AD-related clinical hallmarks.
Journal: Acta neuropathologica communications
Published: May 23, 2024
The amyloid precursor protein and its derived fragments concomitantly contribute to the alterations of mitochondrial transport machinery in Alzheimer's disease.
Journal: Cell death & disease
Published: August 04, 2023
Modifications of the endosomal compartment in fibroblasts from sporadic Alzheimer's disease patients are associated with cognitive impairment.
Journal: Translational psychiatry
Published: February 23, 2022
Mitophagy in Alzheimer's disease: Molecular defects and therapeutic approaches.
Journal: Molecular psychiatry
Published: January 18, 2022
Primary brain cell infection by Toxoplasma gondii reveals the extent and dynamics of parasite differentiation and its impact on neuron biology.
Journal: Open biology
Published: October 05, 2021
Correction to: Alzheimer's genetic risk factor FERMT2 (Kindlin-2) controls axonal growth and synaptic plasticity in an APP-dependent manner.
Journal: Molecular psychiatry
Published: January 05, 2021
Molecular Dysfunctions of Mitochondria-Associated Membranes (MAMs) in Alzheimer's Disease.
Journal: International journal of molecular sciences
Published: November 10, 2020
Accumulation of amyloid precursor protein C-terminal fragments triggers mitochondrial structure, function, and mitophagy defects in Alzheimer's disease models and human brains.
Journal: Acta neuropathologica
Published: June 09, 2020
Last Updated: 09/26/2026