Felix Seyler-Hoppe

Heidelberg, BW, Germany

Last Updated: 09/26/2026

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Found 25 publications

Pleiotropic Effects of Metformin on the Chemotherapy Response of HPV-Positive Cancer Cells.

Pleiotropic Effects of Metformin on the Chemotherapy Response of HPV-Positive Cancer Cells.

Journal: Journal of medical virology

Published: March 20, 2025

Hypoxic HPV-Positive Cancer Cells Are Particularly Sensitive to the Pro-Senescent Effects of B-MYB Repression Due to the Lack of Compensatory A-MYB Induction.

Hypoxic HPV-Positive Cancer Cells Are Particularly Sensitive to the Pro-Senescent Effects of B-MYB Repression Due to the Lack of Compensatory A-MYB Induction.

Journal: Journal of medical virology

Published: February 14, 2025

E6AP is essential for the proliferation of HPV-positive cancer cells by preventing senescence.

E6AP is essential for the proliferation of HPV-positive cancer cells by preventing senescence.

Journal: PLoS pathogens

Published: October 23, 2024

The impact of cycling hypoxia on the phenotype of HPV-positive cervical cancer cells.

The impact of cycling hypoxia on the phenotype of HPV-positive cervical cancer cells.

Journal: Journal of medical virology

Published: August 30, 2023

Revisiting the role of endogenous STAT3 in HPV-positive cervical cancer cells.

Revisiting the role of endogenous STAT3 in HPV-positive cervical cancer cells.

Journal: Journal of medical virology

Published: May 25, 2023

FAM57A (Family with Sequence Similarity 57 Member A) Is a Cell-Density-Regulated Protein and Promotes the Proliferation and Migration of Cervical Cancer Cells.

FAM57A (Family with Sequence Similarity 57 Member A) Is a Cell-Density-Regulated Protein and Promotes the Proliferation and Migration of Cervical Cancer Cells.

Journal: Cells

Published: September 14, 2022

Dickkopf-1 expression is repressed by oncogenic human papillomaviruses (HPVs) and regulates the Cisplatin sensitivity of HPV-positive cancer cells in a JNK-dependent manner.

Dickkopf-1 expression is repressed by oncogenic human papillomaviruses (HPVs) and regulates the Cisplatin sensitivity of HPV-positive cancer cells in a JNK-dependent manner.

Journal: International journal of cancer

Published: April 27, 2022

Delineating the Switch between Senescence and Apoptosis in Cervical Cancer Cells under Ciclopirox Treatment.

Delineating the Switch between Senescence and Apoptosis in Cervical Cancer Cells under Ciclopirox Treatment.

Journal: Cancers

Published: August 19, 2021

Effects of Metformin on the virus/host cell crosstalk in human papillomavirus-positive cancer cells.

Effects of Metformin on the virus/host cell crosstalk in human papillomavirus-positive cancer cells.

Journal: International journal of cancer

Published: December 14, 2020

ITIH5 shows tumor suppressive properties in cervical cancer cells grown as multicellular tumor spheroids.

ITIH5 shows tumor suppressive properties in cervical cancer cells grown as multicellular tumor spheroids.

Journal: American journal of translational research

Published: February 10, 2020

Effects of the antifungal agent ciclopirox in HPV-positive cancer cells: Repression of viral E6/E7 oncogene expression and induction of senescence and apoptosis.

Effects of the antifungal agent ciclopirox in HPV-positive cancer cells: Repression of viral E6/E7 oncogene expression and induction of senescence and apoptosis.

Journal: International journal of cancer

Published: June 19, 2019

PI3K/AKT/mTOR Signaling Regulates the Virus/Host Cell Crosstalk in HPV-Positive Cervical Cancer Cells.

PI3K/AKT/mTOR Signaling Regulates the Virus/Host Cell Crosstalk in HPV-Positive Cervical Cancer Cells.

Journal: International journal of molecular sciences

Published: April 05, 2019

Showing 1-12 of 25

Last Updated: 09/26/2026