Fernando C. Baltanas

Sevilla, AN, Spain

Last Updated: 09/26/2026

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Found 21 publications

SOS1: tracking the evolving path from promising to actionable therapeutic target in RAS-dependent cancers.

SOS1: tracking the evolving path from promising to actionable therapeutic target in RAS-dependent cancers.

Journal: Molecular cancer
Published: January 16, 2026

Cajal's organization of neuronal nucleus revisited.

Cajal's organization of neuronal nucleus revisited.

Journal: Frontiers in neuroanatomy
Published: October 14, 2025

SOS1 inhibitor BI-3406 shows in vivo antitumor activity akin to genetic ablation and synergizes with a KRASG12D inhibitor in KRAS LUAD.

SOS1 inhibitor BI-3406 shows in vivo antitumor activity akin to genetic ablation and synergizes with a KRASG12D inhibitor in KRAS LUAD.

Journal: Proceedings of the National Academy of Sciences of the United States of America
Published: March 12, 2025

Sos1 ablation alters focal adhesion dynamics and increases Mmp2/9-dependent gelatinase activity in primary mouse embryonic fibroblasts.

Sos1 ablation alters focal adhesion dynamics and increases Mmp2/9-dependent gelatinase activity in primary mouse embryonic fibroblasts.

Journal: Cell communication and signaling : CCS
Published: October 24, 2024

Advances in Molecular Research of Oncogenes.

Advances in Molecular Research of Oncogenes.

Journal: International journal of molecular sciences
Published: March 15, 2023

Critical requirement of SOS1 for tumor development and microenvironment modulation in KRASG12D-driven lung adenocarcinoma.

Critical requirement of SOS1 for tumor development and microenvironment modulation in KRASG12D-driven lung adenocarcinoma.

Journal: Nature communications
Published: February 21, 2023

Critical Requirement of SOS1 for Development of BCR/ABL-Driven Chronic Myelogenous Leukemia.

Critical Requirement of SOS1 for Development of BCR/ABL-Driven Chronic Myelogenous Leukemia.

Journal: Cancers
Published: July 19, 2022

The Childhood-Onset Neurodegeneration with Cerebellar Atrophy (CONDCA) Disease Caused by AGTPBP1 Gene Mutations: The Purkinje Cell Degeneration Mouse as an Animal Model for the Study of this Human Disease.

The Childhood-Onset Neurodegeneration with Cerebellar Atrophy (CONDCA) Disease Caused by AGTPBP1 Gene Mutations: The Purkinje Cell Degeneration Mouse as an Animal Model for the Study of this Human Disease.

Journal: Biomedicines
Published: August 16, 2021

SOS2 Comes to the Fore: Differential Functionalities in Physiology and Pathology.

SOS2 Comes to the Fore: Differential Functionalities in Physiology and Pathology.

Journal: International journal of molecular sciences
Published: June 02, 2021

Functional Specificity of the Members of the Sos Family of Ras-GEF Activators: Novel Role of Sos2 in Control of Epidermal Stem Cell Homeostasis.

Functional Specificity of the Members of the Sos Family of Ras-GEF Activators: Novel Role of Sos2 in Control of Epidermal Stem Cell Homeostasis.

Journal: Cancers
Published: March 04, 2021

Critical requirement of SOS1 RAS-GEF function for mitochondrial dynamics, metabolism, and redox homeostasis.

Critical requirement of SOS1 RAS-GEF function for mitochondrial dynamics, metabolism, and redox homeostasis.

Journal: Oncogene
Published: February 01, 2021

SOS GEFs in health and disease.

SOS GEFs in health and disease.

Journal: Biochimica et biophysica acta. Reviews on cancer
Published: July 30, 2020
Showing 1-12 of 21

Last Updated: 09/26/2026