Fiona M. Mccutcheon

Melbourne, VIC, Australia

Last Updated: 09/26/2026

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Found 9 publications

Tenecteplase (TNK) and other TNK formulations lose PAI-1 resistance in their two-chain form: Comparison with alteplase.

Tenecteplase (TNK) and other TNK formulations lose PAI-1 resistance in their two-chain form: Comparison with alteplase.

Journal: Blood advances

Published: April 28, 2026

A Coumarin-Based Array for the Discrimination of Amyloids.

A Coumarin-Based Array for the Discrimination of Amyloids.

Journal: ACS sensors

Published: February 05, 2024

Plasma from patients with vaccine-induced immune thrombotic thrombocytopenia displays increased fibrinolytic potential and enhances tissue-type plasminogen activator but not urokinase-mediated plasminogen activation.

Plasma from patients with vaccine-induced immune thrombotic thrombocytopenia displays increased fibrinolytic potential and enhances tissue-type plasminogen activator but not urokinase-mediated plasminogen activation.

Journal: Journal of thrombosis and haemostasis : JTH

Published: August 20, 2023

Tranexamic acid in a mouse model of cerebral amyloid angiopathy: setting the stage for a novel stroke treatment approach.

Tranexamic acid in a mouse model of cerebral amyloid angiopathy: setting the stage for a novel stroke treatment approach.

Journal: Research and practice in thrombosis and haemostasis

Published: April 10, 2023

Tissue-type plasminogen activator induces conditioned receptive field plasticity in the mouse auditory cortex.

Tissue-type plasminogen activator induces conditioned receptive field plasticity in the mouse auditory cortex.

Journal: iScience

Published: July 05, 2022

Tranexamic acid alters the immunophenotype of phagocytes after lower limb surgery.

Tranexamic acid alters the immunophenotype of phagocytes after lower limb surgery.

Journal: Thrombosis journal

Published: December 14, 2021

Tranexamic acid modulates the cellular immune profile after traumatic brain injury in mice without hyperfibrinolysis.

Tranexamic acid modulates the cellular immune profile after traumatic brain injury in mice without hyperfibrinolysis.

Journal: Journal of thrombosis and haemostasis : JTH

Published: March 03, 2019

Activated CD8+ T Cells Cause Long-Term Neurological Impairment after Traumatic Brain Injury in Mice.

Activated CD8+ T Cells Cause Long-Term Neurological Impairment after Traumatic Brain Injury in Mice.

Journal: Cell reports

Published: December 19, 2017

Selective inhibition of brain endothelial Rho-kinase-2 provides optimal protection of an in vitro blood-brain barrier from tissue-type plasminogen activator and plasmin.

Selective inhibition of brain endothelial Rho-kinase-2 provides optimal protection of an in vitro blood-brain barrier from tissue-type plasminogen activator and plasmin.

Journal: PloS one

Published: January 11, 2017

Showing 1-9 of 9

Last Updated: 09/26/2026