Francesca A. Aiello

Rome, Italy

Last Updated: 09/26/2026

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Found 11 publications

HPV16 recruitment of SMARCAL1 to viral and host replication forks is required for the viral life cycle.

HPV16 recruitment of SMARCAL1 to viral and host replication forks is required for the viral life cycle.

Journal: mBio

Published: March 30, 2026

HPV16 recruitment of SMARCAL1 to viral and host replication forks is required for the viral life cycle.

HPV16 recruitment of SMARCAL1 to viral and host replication forks is required for the viral life cycle.

Journal: bioRxiv : the preprint server for biology

Published: August 08, 2025

The HIV Protease Inhibitor Ritonavir Reverts the Mesenchymal Phenotype Induced by Inflammatory Cytokines in Normal and Tumor Oral Keratinocytes to an Epithelial One, Increasing the Radiosensitivity of Tumor Oral Keratinocytes.

The HIV Protease Inhibitor Ritonavir Reverts the Mesenchymal Phenotype Induced by Inflammatory Cytokines in Normal and Tumor Oral Keratinocytes to an Epithelial One, Increasing the Radiosensitivity of Tumor Oral Keratinocytes.

Journal: Cancers

Published: June 24, 2025

Neddylation inhibition induces DNA double-strand breaks, hampering tumor growth in vivo, and promotes radiosensitivity in PAX3-FOXO1 rhabdomyosarcoma.

Neddylation inhibition induces DNA double-strand breaks, hampering tumor growth in vivo, and promotes radiosensitivity in PAX3-FOXO1 rhabdomyosarcoma.

Journal: Cell death discovery

Published: April 18, 2025

HDAC3 genetic and pharmacologic inhibition radiosensitizes fusion positive rhabdomyosarcoma by promoting DNA double-strand breaks.

HDAC3 genetic and pharmacologic inhibition radiosensitizes fusion positive rhabdomyosarcoma by promoting DNA double-strand breaks.

Journal: Cell death discovery

Published: February 07, 2024

A double-ring of human RAD52 remodels replication forks restricting fork reversal.

A double-ring of human RAD52 remodels replication forks restricting fork reversal.

Journal: bioRxiv : the preprint server for biology

Published: November 28, 2023

The RAD52 double-ring remodels replication forks restricting fork reversal.

The RAD52 double-ring remodels replication forks restricting fork reversal.

Journal: Nature

Published: November 14, 2023

SHP2's gain-of-function in Werner syndrome causes childhood disease onset likely resulting from negative genetic interaction.

SHP2's gain-of-function in Werner syndrome causes childhood disease onset likely resulting from negative genetic interaction.

Journal: Clinical genetics

Published: February 17, 2022

Author Correction: Rad52 prevents excessive replication fork reversal and protects from nascent strand degradation.

Author Correction: Rad52 prevents excessive replication fork reversal and protects from nascent strand degradation.

Journal: Nature communications

Published: May 03, 2019

RAD51 and mitotic function of mus81 are essential for recovery from low-dose of camptothecin in the absence of the WRN exonuclease.

RAD51 and mitotic function of mus81 are essential for recovery from low-dose of camptothecin in the absence of the WRN exonuclease.

Journal: Nucleic acids research

Published: August 29, 2018

Rad52 prevents excessive replication fork reversal and protects from nascent strand degradation.

Rad52 prevents excessive replication fork reversal and protects from nascent strand degradation.

Journal: Nature communications

Published: August 05, 2018

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Last Updated: 09/26/2026