Frederique F. Cornelis
Leuven, VLG, Belgium
Last Updated: 09/26/2026
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Found 14 publications
EXTL3 dysfunction identified as a driver of aberrant bone development in severe familial ankylosing spondylitis.
Journal: Annals of the rheumatic diseases
Published: March 31, 2026
Temporal profiling of lipid mediators in synovium and tibial plateau during joint inflammation in a collagenase-induced mouse model of osteoarthritis.
Journal: Osteoarthritis and cartilage
Published: March 07, 2025
Lipidomics unravels lipid changes in osteoarthritis articular cartilage.
Journal: Annals of the rheumatic diseases
Published: October 04, 2024
IGF1 drives Wnt-induced joint damage and is a potential therapeutic target for osteoarthritis.
Journal: Nature communications
Published: March 21, 2024
The sacroiliac joint: An original and highly sensitive tool to highlight altered bone phenotype in murine models of skeletal disorders.
Journal: Bone
Published: May 11, 2023
Osteomodulin downregulation is associated with osteoarthritis development.
Journal: Bone research
Published: December 28, 2022
Inhibition of KDM7A/B histone demethylases restores H3K79 methylation and protects against osteoarthritis.
Journal: Annals of the rheumatic diseases
Published: December 19, 2022
Inhibiting thyroid activation in aged human explants prevents mechanical induced detrimental signalling by mitigating metabolic processes.
Journal: Rheumatology (Oxford, England)
Published: October 20, 2021
ANP32A regulates ATM expression and prevents oxidative stress in cartilage, brain, and bone.
Journal: Science translational medicine
Published: December 22, 2017
DOT1L safeguards cartilage homeostasis and protects against osteoarthritis.
Journal: Nature communications
Published: November 14, 2016
Aberrant Calreticulin Expression in Articular Cartilage of Dio2 Deficient Mice.
Journal: PloS one
Published: January 14, 2016
The effect of forced exercise on knee joints in Dio2(-/-) mice: type II iodothyronine deiodinase-deficient mice are less prone to develop OA-like cartilage damage upon excessive mechanical stress.
Journal: Annals of the rheumatic diseases
Published: September 11, 2014
Last Updated: 09/26/2026