Fugen Shangguan

Zhejiang Key Laboratory Of Intelligent Cancer Biomarker Discovery And Translation
Wenzhou, 325035, China

Last Updated: 09/26/2026

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Found 24 publications

Corrigendum to "Targeting GSS-mediated glutathione synthesis: 6-Met induces oxidative stress dependent apoptosis via triggering mitochondrial bioenergetics metabolism crisis and mTOR suppression in glioblastoma" [Redox Biol. 96 (2026) 104334].

Corrigendum to "Targeting GSS-mediated glutathione synthesis: 6-Met induces oxidative stress dependent apoptosis via triggering mitochondrial bioenergetics metabolism crisis and mTOR suppression in glioblastoma" [Redox Biol. 96 (2026) 104334].

Journal: Redox biology

Published: August 28, 2026

Targeting GSS-mediated glutathione synthesis: 6-Met induces oxidative stress dependent apoptosis via triggering mitochondrial bioenergetics metabolism crisis and mTOR suppression in glioblastoma.

Targeting GSS-mediated glutathione synthesis: 6-Met induces oxidative stress dependent apoptosis via triggering mitochondrial bioenergetics metabolism crisis and mTOR suppression in glioblastoma.

Journal: Redox biology

Published: April 07, 2026

SLC40A1-mediated positive feedback loop with M1 macrophages suppresses epithelial ovarian cancer progression.

SLC40A1-mediated positive feedback loop with M1 macrophages suppresses epithelial ovarian cancer progression.

Journal: Frontiers in immunology

Published: September 20, 2025

Demethoxycurcumin induces metabolic crisis and ATF4/ATF3/CHOP-dependent cell death in hepatocellular carcinoma.

Demethoxycurcumin induces metabolic crisis and ATF4/ATF3/CHOP-dependent cell death in hepatocellular carcinoma.

Journal: Chemico-biological interactions

Published: February 05, 2025

Fucoxanthin suppresses pancreatic cancer progression by inducing bioenergetics metabolism crisis and promoting SLC31A1‑mediated sensitivity to DDP.

Fucoxanthin suppresses pancreatic cancer progression by inducing bioenergetics metabolism crisis and promoting SLC31A1‑mediated sensitivity to DDP.

Journal: International journal of oncology

Published: September 06, 2024

LonP1 Links Mitochondria-ER Interaction to Regulate Heart Function.

LonP1 Links Mitochondria-ER Interaction to Regulate Heart Function.

Journal: Research (Washington, D.C.)

Published: March 17, 2023

A novel mechanism of 6-methoxydihydroavicine in suppressing ovarian carcinoma by disrupting mitochondrial homeostasis and triggering ROS/ MAPK mediated apoptosis.

A novel mechanism of 6-methoxydihydroavicine in suppressing ovarian carcinoma by disrupting mitochondrial homeostasis and triggering ROS/ MAPK mediated apoptosis.

Journal: Frontiers in pharmacology

Published: November 09, 2022

A novel mechanism of cannabidiol in suppressing ovarian cancer through LAIR-1 mediated mitochondrial dysfunction and apoptosis.

A novel mechanism of cannabidiol in suppressing ovarian cancer through LAIR-1 mediated mitochondrial dysfunction and apoptosis.

Journal: Environmental toxicology

Published: October 14, 2022

N-glycosylation stabilizes MerTK and promotes hepatocellular carcinoma tumor growth.

N-glycosylation stabilizes MerTK and promotes hepatocellular carcinoma tumor growth.

Journal: Redox biology

Published: May 01, 2022

Effects of natural 24-epibrassinolide on inducing apoptosis and restricting metabolism in hepatocarcinoma cells.

Effects of natural 24-epibrassinolide on inducing apoptosis and restricting metabolism in hepatocarcinoma cells.

Journal: Phytomedicine : international journal of phytotherapy and phytopharmacology

Published: February 13, 2022

6-methoxydihydroavicine, the alkaloid extracted from Macleaya cordata (Willd.) R. Br. (Papaveraceae), triggers RIPK1/Caspase-dependent cell death in pancreatic cancer cells through the disruption of oxaloacetic acid metabolism and accumulation of reactive oxygen species.

6-methoxydihydroavicine, the alkaloid extracted from Macleaya cordata (Willd.) R. Br. (Papaveraceae), triggers RIPK1/Caspase-dependent cell death in pancreatic cancer cells through the disruption of oxaloacetic acid metabolism and accumulation of reactive oxygen species.

Journal: Phytomedicine : international journal of phytotherapy and phytopharmacology

Published: February 07, 2022

Synthetic oleanane triterpenoid derivative CDDO-Me disrupts cellular bioenergetics to suppress pancreatic ductal adenocarcinoma via targeting SLC1A5.

Synthetic oleanane triterpenoid derivative CDDO-Me disrupts cellular bioenergetics to suppress pancreatic ductal adenocarcinoma via targeting SLC1A5.

Journal: Journal of biochemical and molecular toxicology

Published: May 06, 2021

Showing 1-12 of 24

Last Updated: 09/26/2026