Gagan B. Panigrahi

Toronto, ON M5G0A4, Canada

Last Updated: 09/26/2026

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Found 12 publications

Interventionally targeting somatic CAG expansions can be a rapid disease-modifying therapeutic avenue: Preclinical evidence.

Interventionally targeting somatic CAG expansions can be a rapid disease-modifying therapeutic avenue: Preclinical evidence.

Journal: bioRxiv : the preprint server for biology
Published: May 07, 2025

Antagonistic roles of canonical and Alternative-RPA in disease-associated tandem CAG repeat instability.

Antagonistic roles of canonical and Alternative-RPA in disease-associated tandem CAG repeat instability.

Journal: Cell
Published: June 30, 2022

FAN1 exo- not endo-nuclease pausing on disease-associated slipped-DNA repeats: A mechanism of repeat instability.

FAN1 exo- not endo-nuclease pausing on disease-associated slipped-DNA repeats: A mechanism of repeat instability.

Journal: Cell reports
Published: May 06, 2021

FAN1, a DNA Repair Nuclease, as a Modifier of Repeat Expansion Disorders.

FAN1, a DNA Repair Nuclease, as a Modifier of Repeat Expansion Disorders.

Journal: Journal of Huntington's disease
Published: February 13, 2021

Functional Repair Assay for the Diagnosis of Constitutional Mismatch Repair Deficiency From Non-Neoplastic Tissue.

Functional Repair Assay for the Diagnosis of Constitutional Mismatch Repair Deficiency From Non-Neoplastic Tissue.

Journal: Journal of clinical oncology : official journal of the American Society of Clinical Oncology
Published: January 05, 2019

A slipped-CAG DNA-binding small molecule induces trinucleotide-repeat contractions in vivo.

A slipped-CAG DNA-binding small molecule induces trinucleotide-repeat contractions in vivo.

Journal: Nature genetics
Published: October 20, 2018

Absence of MutSβ leads to the formation of slipped-DNA for CTG/CAG contractions at primate replication forks.

Absence of MutSβ leads to the formation of slipped-DNA for CTG/CAG contractions at primate replication forks.

Journal: DNA repair
Published: December 01, 2015

Combined hereditary and somatic mutations of replication error repair genes result in rapid onset of ultra-hypermutated cancers.

Combined hereditary and somatic mutations of replication error repair genes result in rapid onset of ultra-hypermutated cancers.

Journal: Nature genetics
Published: October 30, 2014

An infant with MLH3 variants, FOXG1-duplication and multiple, benign cranial and spinal tumors: A clinical exome sequencing study.

An infant with MLH3 variants, FOXG1-duplication and multiple, benign cranial and spinal tumors: A clinical exome sequencing study.

Journal: Genes, chromosomes & cancer
Published: October 16, 2014

Processing of double-R-loops in (CAG)·(CTG) and C9orf72 (GGGGCC)·(GGCCCC) repeats causes instability.

Processing of double-R-loops in (CAG)·(CTG) and C9orf72 (GGGGCC)·(GGCCCC) repeats causes instability.

Journal: Nucleic acids research
Published: August 23, 2014

Mismatch repair genes Mlh1 and Mlh3 modify CAG instability in Huntington's disease mice: genome-wide and candidate approaches.

Mismatch repair genes Mlh1 and Mlh3 modify CAG instability in Huntington's disease mice: genome-wide and candidate approaches.

Journal: PLoS genetics
Published: April 03, 2013

Human mismatch repair protein hMutLα is required to repair short slipped-DNAs of trinucleotide repeats.

Human mismatch repair protein hMutLα is required to repair short slipped-DNAs of trinucleotide repeats.

Journal: The Journal of biological chemistry
Published: October 23, 2012
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Last Updated: 09/26/2026